John Carriero has argued that for Spinoza there is no final causality in the Aristotelian sense and that the striving of things is merely to be understood in terms of metaphysical inertia. This paper makes a case against this claim. First it is argued that Spinoza’s notion of striving does in principle meet Thomas Aquinas’ criterion for final causation. Second it is shown that Carriero’s denial of final causation in Spinoza leads to a deflationary interpretation (...) of Spinoza’s notions of the good and striving of things, which is at odds with many passages of Spinoza’s Ethics. One can only do justice to these passages if one assumes that Spinoza did accept final causation in the traditional sense. John Carriero argumentierte, dass Spinoza keine finale Kausalität im aristotelischen Sinne vertrete, sondern das Streben von Dingen als Ausdruck einer Art metaphysischen Trägheit verstehe. Dagegen, so wird hier ausgeführt, sprechen zwei Gründe: Erstens genügt Spinozas Charakterisierung des Strebens von Dingen prinzipiell Thomas von Aquins Definition finaler Verursachung. Zweitens führt Carrieros Zurückweisung finaler Verursachung zu einer deflationären Lesart von Spinozas Begriffen des Guten und des Strebens von Dingen, die mit vielen Passagen aus Spinozas Ethik unvereinbar sind. Diesen Passagen kann nur Rechnung getragen werden, wenn man annimmt, dass Spinoza finale Verursachung im traditionellen Sinn akzeptierte. (shrink)

Early modern philosophers rejected various important aspects of Aristotelianism. Current scholarship debates the question to what extent the early moderns rejected final causation. Leibniz explicitly endorsed it. I argue that his notion of final causation should be understood in connection with his resurrection of substantial forms and his seeing such forms on the model of the soul. I relate Leibniz’ conception of final causation to the Aristotelian background as well as Descartes’s treatment of teleology. (...) I argue that he agreed with a view found in Aristotelianism that genuine efficiently causal powers require immanent teleology and with Descartes that immanent teleology presupposes cognition in the agent. (shrink)

In semiotics, final causation can be related to the process of determination (PAPE, 1993). From Peirce’s point of view, determination is not a causal determinism, but a delimitation of a range of possibilities. One starts from objects towards interpretants, in a process mediated by the sign, in which the dynamic object works as a force that constrains interpretants to correspond to their objects. The correspondence between object and interpretant is important because it is through a generated interpretant that (...) the object of a sign can be known. Even though this process of determination coincides with the idea of final causation, there is a certain indeterminacy in it. For Peirce (EP 2:353, 1905), vagueness and generality are two types of indeterminacy. In the terms of the phenomenological categories, vagueness is an indeterminacy of the order of firstness, generality an indeterminacy of the order of thirdness, and both, to some extent, are opposed to that which is defined, which belongs to secondness. Each aspect of the sign may vary according to the three phenomenological categories. Consequently, degrees of imprecision are added to the semiotic process, which is a determination process. Peirce asserts that the perfect precision of thought is theoretically unattainable (SS 11, 1903). Every sign is vague or general at least to some degree. In this paper, we seek to perceive degrees of indetermination and causality from an analysis of the kinds of objects and interpretants proposed by Peirce in the system of 28 sign classes. (shrink)

ABSTRACT: With a view to suggesting the possible relevance of Aristotelian thought to current notions of complexity and self-organization, studies Aristotlenard cells, and the theories of Schneider, Kay, and D. Sagan.

The article examines how Reimarus reorients concepts borrowed from Leibniz and Wolff – the principles of perfection, harmony and continuity – in order to feed his own natural religion project. Teleology is understood as a doctrine aiming at proving not only God’s perfections, but also the effects of the divine wisdom on creatures. Consequently, recourse to final causes in natural philosophy cannot remain at the level of general reasons, as Maupertuis’s principle of least action does, but rather ought to (...) be used in the explanation of the specific purpose of beings. (shrink)

For many areas of philosophy, we lack an understanding of their developments between the fourteenth and seventeenth centuries. One such area is the development of the notion of final causation. The rejection of final causation is often described as one of the distinguishing hallmarks of so called Early Modern philosophy in relation to the Scholastic philosophical tradition. Our lack of understanding of the development of this notion in philosophy therefore impedes our ability to write an adequate (...) history of philosophy spanning these centuries. In this article, the notion of final causation as treated in the works of Chrysostom Javellus (1472–1538) and Francis Silvestri (of Ferrara) (1474–1526) is presented. It is argued that the treatment of final causation in these thinkers is already shaped by concerns regarding finality that we find in Early Modern philosophy. (shrink)

According to a standard picture of Leibniz’s mature views on creaturely causation, Leibniz held what some interpreters have described as his ‘thesis of spontaneity’: “every non-initial, nonmiraculous state of every created substance has as a real cause some preceding state of that very substance.”2 Evidence for this thesis is abundantly available throughout Leibniz’s mature work and here are some prominent instances.

This paper revives the idea of final cause as a way of considering how the meaning of occurrences--what they have permitted in our lives, as opposed to what produced them or how they fit into a prior plan--is essential to our understanding them.

In “The Fixation of Belief,” Peirce apparently accepts the causal claim that real physical objects cause us to reach an indefeasible “final opinion” concerning them. In “How to Make Our Ideas Clear,” he apparently accepts the constitutive claim that for physical objects to be real just is for them to be represented in that opinion. These claims initially seem inconsistent, since causal claims are explanatory and since equivalent claims cannot explain one another. Contrary to prominent suggestions that Peirce rejected (...) the constitutive claim, however, he actually accepted both, reconciling them via a Humean denial that causal claims are genuinely explanatory. (shrink)

The world contains objective causal relations and universals, both of which are intimately connected. If these claims are true, they must have far-reaching consequences, breathing new life into the theory of empirical knowledge and reinforcing epistemological realism. Without causes and universals, Professor Fales argues, realism is defeated, and idealism or scepticism wins. Fales begins with a detailed analysis of David Hume's argument that we have no direct experience of necessary connections between events, concluding that Hume was mistaken on this fundamental (...) point. Then, adopting the view of Armstrong and others that causation is grounded in a second-order relation between universals, he explores a range of topics for which the resulting analysis of causation has systematic implications. In particular, causal identity conditions for physical universals are proposed, which generate a new argument for Platonism. The nature of space and time is discussed, with arguments against backward causation and for the view that space and time can exist independently of matter or causal process. Many of Professor Fales's conclusions seem to run counter to received opinion among contemporary empiricists. Yet his method is classically empiricist in spirit, and a chief motive for these metaphysical explorations is epistemological. The final chapters investigate the perennial question of whether an empiricist, internalist and foundational epistemology can support scientific realism. (shrink)

The paper builds on the basically Humean idea that A is a cause of B iff A and B both occur, A precedes B, and A raises the metaphysical or epistemic status of B given the obtaining circumstances. It argues that in pursuit of a theory of deterministic causation this ‘status raising’ is best explicated not in regularity or counterfactual terms, but in terms of ranking functions. On this basis, it constructs a rigorous theory of deterministic causation that (...) successfully deals with cases of overdetermination and pre-emption. It finally indicates how the account's profound epistemic relativization induced by ranking theory can be undone. Introduction Variables, propositions, time Induction first Causation Redundant causation Objectivization. (shrink)

Mental causation is notoriously threatened by the causal exclusion argument. A prominent strategy to save mental causation from causal exclusion consists in subscribing to an interventionist account of causation. This move has, however, recently been challenged by several authors. In this paper, we do two things: We (i) develop what we consider to be the strongest version of the interventionist causal exclusion argument currently on the market and (ii) propose a new way how it can in principle (...) be overcome. In particular, we propose to replace strict supervenience in the assumption that the mental supervenes on the physical by probabilistic supervenience and show how this move has the potential to license the inference to mental causation. Finally, we argue that probabilistic supervenience captures some of the most important intuitions that strict supervenience captures and discuss possible objections to weakening strict supervenience in the way we suggest. (shrink)

Downward causation exercised by emergent properties of wholes upon their lower-level constituents’ properties has been accused of conceptual and metaphysical incoherence. Only upward causation is usually peacefully accepted. The aim of this paper is to criticize and refuse the traditional hierarchical-vertical way of conceiving both types of causation, although preserving their deepest ontological significance, as well as the widespread acceptance of the traditional atomistic-combinatorial view of the entities and the relations that constitute the so-called ‘emergence base’. Assuming (...) those two perspectives with no reserves, we are condemned to confine our debate to the question of whether reified wholes can have the power to downwardly change or influence their lower-level parts, a question which seems profoundly misleading to me. I therefore propose an alternative relational ontological view, assuming a straightforward horizontal and intra-level way of representing those putative cases of cross-level causation. I finally confront two recent replies to Kim’s well-known objections to DC—Craver and Bechtel and Kistler :595–609, 2009)—, emphasizing their global positive approaches, as well as the reasons why their accounts still seem insufficient to me. I conclude arguing that both Kim’s principle of the causal closure of the physical domain and its allegation of an overdetermination in cases of DC can be surpassed by the new relational ontological perspective presented here. (shrink)

I defend the thesis that, if there are substances, substance causation (i.e., causation by substances) is the only sort of causation in the universe – or the only fundamental sort. Subsequently, I develop an account of substance causation that is partly grounded on a peculiar interpretation of absolute change (i.e., of entities' coming and ceasing to be) and qualitative change, on some ontological assumptions about modes (i.e., individual properties that ontologically depend on their bearers) and powers. (...) Finally, I reply to some objections against substance causation in general (e.g., C. D. Broad's objection against causes' being non-essentially-dated entities) and to some possible objections against my own account and I briefly explore three advantages given by the acceptance of the latter. (shrink)

In this article I examine the relation between causation and moral responsibility. I distinguish four possible views about that relation. One is the standard view: the view that an agent's moral responsibility for an outcome requires, and is grounded in, the agent's causal responsibility for it. I discuss several challenges to the standard view, which motivate the three remaining views. The final view – the view I argue for – is that causation is the vehicle of transmission (...) of moral responsibility. According to this view, although moral responsibility does not require causation, causation still grounds moral responsibility. (shrink)

Reasoning about causation in fact is an essential element of attributing legal responsibility. Therefore, the automation of the attribution of legal responsibility requires a modelling effort aimed at the following: a thorough understanding of the relation between the legal concepts of responsibility and of causation in fact; a thorough understanding of the relation between causation in fact and the common sense concept of causation; and, finally, the specification of an ontology of the concepts that are minimally (...) required for (automatic) common sense reasoning about causation. This article offers a worked-out example of the indicated analysis. Such example consists of: a definition of the legal concept of responsibility (in terms of liability and accountability); a definition of the legal concept of causation in fact (in terms of the initiation of physical processes by an agent and of the provision of reasons and/or opportunities to other agents); CausatiOnt, an AI-like ontology of the common sense (causal) concepts that are minimally needed for reasoning about the legal concept of causation in fact (in particular, the concepts of category, dimension, object, agent, process, event and act). (shrink)

Quentin Smith’s idea is that God being the originating cause of the universe is logically inconsistent with all extant definitions of causation, and thus logically impossible. Thus, for example the God of the Philosophers couldn’t have created the Universe, not even in both its senses, in both literal and analogical senses. The thesis is advanced by accounts of the usual views of “cause”. It is maintained these is successful. Such I shall then offer an account of divine causation (...) of my own, and thus attempt to argue that Smith has not shown that the relation that God has to the universe is not a causal relation. Such as a Humean or that of David Lewis sense and of the “singularist” view of C. J. Ducasse would fail the analogical. And Malebranche’s “occasionalism” is surely an exception. If we turn to the other kind then it seems to be a case of “if the data are analogical-in, then the data will be that too”. Finally, it is argued that it is more productive to consider particular individual theistic powers and perfections, for these are mongrels which literality and of analogy are compounded. (shrink)

Agent causation presupposes that actions are behaviors under the causal control of the agent’s mental states, its beliefs and desires. Here the idea of conscious causation in causal explanations of actions is examined, specifically, actions said to be the result of conscious efforts. Causal–functionalist theories of consciousness purport to be naturalistic accounts of the causal efficacy of consciousness. Flanagan argues that his causal–functionalist theory of consciousness satisfies naturalistic constraints on causation and that his causal efficacy thesis is (...) compatible with results of Libet’s experiments on conscious causation. First, the notions of conscious effort and conscious causation are analyzed with respect to the project of naturalizing the mind, that is, the attempt to assimilate folk-psychological explanation to the causal model of explanation in the natural sciences. It is argued that a serious obstacle for any naturalist program is that mental states are individuated by their semantic content, not the mechanistic, physical properties of their neural state instantiations. In particular, it is argued that explanation by reference to mental state content yields not a causal but an interpretive or rationalizing account of action in which the question of causal efficacy is irrelevant. Then, Flanagan’s causal–functionalist theory of consciousness is critically assessed; specifically his interpretations of Libet’s negative experimental results on the causal efficacy of consciousness are diagnosed and disputed. It is contended that Flanagan misinterprets the results of Libet’s consciousness experiments and that his functionalist concept of consciousness fails to yield an adequate explanation of the alleged causal efficacy of consciousness. Finally, his thesis is countered with other experimental results that appear to favor an epiphenomenalist view over the causal efficacy account of consciousness. (shrink)

In recent years, a number of philosophers have attempted to fix paradoxes of the counterfactual account of causation by making causation contrastive. In this framework, causation is understood to be not a two-place relationship between a cause and an effect but a three or four-place relationship between a cause, an effect and a contrast on the side of the cause, the effect or both. I argue that contrasting helps resolving certain paradoxes only if an account of admissibility (...) of the chosen set of contrasts is given. I show by means of numerous examples that it is contextual features that determine admissibility. This way, context becomes part of the semantics of causation. I finally argue that once contextualised, explicit contrasting is redundant: causation is therefore a three-place relationship between a cause, an effect and a context. (shrink)

Downward causation is a widespread and problematic phenomenon. It is typically defined as the causation of lower-level effects by higher-level entities. Downward causation is widespread, as there are many examples of it across different sciences: a cell constraints what happens to its own constituents; a body regulates its own processes; two atoms, when they are appropriately related, make it the case that their own electrons are distributed in certain ways. However, downward causation is also problematic. Roughly, (...) it seems to be at odds with specific scientific and/or epistemological desiderata: first and foremost, that everything can be reduced (one day or another) to the fundamental, micro-physical constituents and goings-on of the universe, so as to provide a unified explanation of everything and a unification of all the sciences " from the bottom ". Indeed, downward causation (if it is an irreducible phenomenon) introduces special causings not only at the higher levels, but also at the lower ones: if, in principle, we cannot fully understand what happens to the electrons without paying attention to the atoms (at the higher level) and we cannot fully understand what happens to the atoms by only paying attention to the electrons (at the lower level), there is no fully lower-level explanation for both higher-level and lower-level goings-on. In this introduction, we shall try to describe the prospects for downward causation in metaphysics and the philosophy of science. After having delved into the connections between downward causation, emergence and levels (§1), we shall discuss the irreducibility of downward causation (§2). We shall then briefly consider how specific metaphysical and epistemological assumptions bear on our understanding of downward causation and of its possibility (§3) and describe some views according to which downward causation is actually non-causal (or it is a special causal relation) (§4). We shall also mention some problems for the connection between downward causation and mental causation (§5) and some scientific examples of downward causation (§6). Finally, we shall summarize the contents of the contributions in this book (§7). (shrink)

Leibniz holds that created substances do not causally interact with each other but that there is causal activity within each such creature. Every created substance constantly changes internally, and each of these changes is caused by the substance itself or by its prior states. Leibniz describes this kind of intra-substance causation both in terms of final causation and in terms of efficient causation. How exactly this works, however, is highly controversial. I will identify what I take (...) to be the major interpretive issues surrounding Leibniz's views on causation and examine several influential interpretations of these views. In ‘Leibniz on Causation – Part 2’ I will then take a closer look at final causation. (shrink)

In many biological experiments, due to gene-redundancy or distributed backup mechanisms, there are no visible effects on the functionality of the organism when a gene is knocked out or down. In such cases there is apparently no counterfactual dependence between the gene and the phenotype in question, although intuitively the gene is causally relevant. Due to relativity of causal relations to causal models, we suggest that such cases can be handled by changing the resolution of the causal model that represents (...) the system. By decreasing the resolution of our causal model, counterfactual dependencies can be established at a higher level of abstraction. By increasing the resolution, stepwise causal dependencies of the right kind can serve as a sufficient condition for causal relevance. Finally, we discuss how introducing a temporal dimension in causal models can account for causation in cases of non-modular systems dynamics. (shrink)

This article aims to provide a brief overview of mental causation problem and its current proposed solutions. Indeed, mental causation turns out as one of the most difficult philosophical conundrums in contemporary philosophy of mind. In the first two sections, we offer an outline of the problem and the philosophical debate about it, and show that mental causation problem is pivotal within the contemporary philosophy of mind. In the third section, we focus on the most popular models (...) of mental causation, namely Kim's and Davidson's accounts, also discussing the objections raised against them. In the final section, we take into consideration some recent proposals poised to solve the mental causation problem, including powerism. Given the logical and metaphysical plausibility of almost all these different options, our conclusion is that mental causation is still an open problem and it is far from being resolved. (shrink)

D. Hume's analysis of causation is critically analyzed in light of certain assumptions that he made regarding the classical Aristotelian causes. Using his widely cited analysis of billiard balls colliding and moving about as an example of how efficient causation is supposedly learned, the argument is made that Hume has overlooked the functioning of final causation in this learning. Thus, in order to understand how a learner might reason back from the presumed "effect" to the "cause" (...) in efficient causation, we must appreciate that the learner is actually employing a final-cause process of predication-that is, of reasoning "for the sake of" precedents extending sequaciously to targets. As final causation underwrites telic description, it is concluded that there is an unrecognized teleology in Hume's analysis of causation. 2012 APA, all rights reserved). (shrink)

This dissertation begins by addressing the question of when a causal model is apt for deciding questions of actual causation with respect to some target situation. I first provide relevant background about causal models, explain what makes them promising as a tool for analyzing actual causation, and motivate the need for a theory of aptness as part of such an analysis (Chapter 1). I then define what it is for a model on a given interpretation to be accurate (...) of, that is, say only true things about, some target situation. This involves a systematization of various representational principles mentioned and/or discussed throughout the literature into a method of interpretation, which I propose be taken as standard (Chapter 2). Next, I explain and address two reasons for which accuracy as I’ve defined it is insufficient for aptness. The first reason – already discussed in the literature – is the problem of structural isomorphs. In response, I propose the aptness condition of Explicit Partial Mediation (Chapter 3). The second reason – which has yet to be noticed – is the problem of the indeterminacy of accuracy. As I demonstrate, a model is accurate of a target situation only relative to a set of background possibilities – what I call a modal profile. It follows that a model represents a situation only relative to some modal profile or other. I go on to discuss the ramifications of this observation for a theory of actual causation in terms of models. I argue that the relativity be taken at face value and built into our metaphysical account of causation, resulting in a view that I call causal relativism (Chapter 4). I explore one advantage of this view in detail: that the resulting account can defend the principle of strong proportionality against several objections (Chapter 5). Finally, I apply the earlier discussion of aptness to attempts to provide a semantics of counterfactuals in terms of causal models – an interventionist semantics. I show how just as a similarity semantics relies on an opaque notion of similarity, an interventionist semantics relies on an analogous notion of aptness. The challenge of articulating aptness thus undermines the claim that an interventionist semantics avoids representational problems inherent in a similarity semantics (Chapter 6). I close with a recap and suggestions for future research (Chapter 7). -/- . (shrink)

I present an alternative account of causation in the biomedical and social sciences according to which the meaning of causal claims is given by their inferential relations to other claims. Specifically, I will argue that causal claims are inferentially related to certain evidential claims as well as claims about explanation, prediction, intervention and responsibility. I explain in some detail what it means for a claim to be inferentially related to another and finally derive some implication of the proposed account (...) for the epistemology, semantics and metaphysics of causation. (shrink)

Twenty years ago Stephen Yablo developed his original theory of mental causation, which has drawn much attention ever since. By providing a detailed reconstruction of Yablo’s approach, this paper first demonstrates that a certain line of critique that has repeatedly been brought forward against Yablo over the last two decades misconstrues the core idea of the model. At the same time, the reconstruction reveals that Yablo’s approach is probably the first explicit version of the “new compatibilism” within the philosophy (...) of mind. This fairly young family of theories essentially claims a non-identity as well as a non-distinctness of mental and physical phenomena. The second part of the paper then demonstrates that the new compatibilist approaches in general, and Yablo’s theory in particular, even though they can resist much of the popular criticism, fall prey to a new theoretical trilemma once the nature of the respective analysantia is made explicit. Finally, a model of the psychophysical relation is developed that may allow the new compatibilists to escape the mentioned trilemma. (shrink)

The central argument of this paper is that compatibilist theories that understand human agent causation as self-determination are consistent with, and can accommodate, important insights from evolutionary and developmental psychology. Agent causation is nothing more than the non-mysterious self-determining capability of persons, understood as embodied, emergent ontological entities whose nature is not fixed due to their uniquely evolved and developed capabilities of language use, cultural construction, self-consciousness and self-understanding, and moral concern. Relevant arguments of Dennett and Searle are (...) adapted to fit within the author's own emergentist, developmental theorizing that treats the self-determining capability of persons as arising within relevant biophysical and sociocultural contexts without being reducible to these contexts. Persons are self-determining in ways that require explanation not only in terms of materialistic efficient causal explanations appropriate to our evolved biophysical nature, but also in terms of final purposive explanations appropriate to our emergence as culture- capable, self-reflective agents, and formal explanations consistent with our evolved and developed nature as particular bio-cultural hybrids. (shrink)

The counterfactual and regularity theories are universal accounts of causation. I argue that these should be generalized to produce local accounts of causation. A hallmark of universal accounts of causation is the assumption that apparent variation in causation between locations must be explained by differences in background causal conditions, by features of the causal-nexus or causing-complex. The local account of causation presented here rejects this assumption, allowing for genuine variation in causation to be explained (...) by differences in location. I argue that local accounts of causation are plausible, and have pragmatic, empirical and theoretical advantages over universal accounts. I then report on the use of presheaves as models of local causation. The use of presheaves as models of local variation has precedents in algebraic geometry, category theory and physics; they are here used as models of local causal variation. The paper presents this idea as stemming from an approach using presheaves as models of local truth. Finally, I argue that a proper balance between universal and local causation can be assuaged by moving from presheaves to fully-fledged sheaf models. (shrink)

There are philosophers who deny that causal relations actually exist in nature, arguing that they are merely a product of our perspective as beings capable of intentional actions. In this paper I briefly explain this thesis and consider that it needs to be complemented with a basic non-causal ontological perspective whichcan account for phenomena taken as causal; I then describe what seems to be a good candidate for such an ontology and finally conclude, however, that it cannot dispense with the (...) concept of causation and that therefore is not the kind of ontology that anti-realism about causal relations requires.Hay filósofos que niegan la existencia de relaciones causales en la naturaleza argumentando que no son sino producto de nuestra perspectiva como seres capaces de acciones intencionales. En este artículo expongo brevemente esta tesis y sostengo que debe contar con el complemento de una ontología no causal básica que dé razón de los fenómenos que se consideran causales. Luego describo lo que parece ser una buena candidata para tal ontología y concluyo que, sin embargo, ésta no puede prescindir del concepto de causalidad y que, por tanto, no es el tipo de ontología que necesita la mencionada tesis antirrealista respecto a la causalidad. (shrink)

In this paper I argue that economic theories have never been able to provide a coherent explanation of the causation requirement in tort law. The economic characterization of this requirement faces insurmountable difficulties, because discourse on tort liability cannot be reduced to a cost-benefit analysis without a loss of meaning. More seriously, I try to show that by describing causation in economic terms, economic theories offer an image of the practice in which the participants incur in logical contradictions (...) and develop patterns of inference that are far from intuitive. For this reason, efficiency cannot be the fundamental principle underlying tort law. Finally, I suggest that economic analysis of law can provide a genuine explanation of certain aspects of legal practice if it relinquishes its reductionist claims. (shrink)

Holtby, Allen and Fairchild are both recent and revolutionary decisions that address an important aspect of the indeterminate causation problem that frequently arises in tort litigation. In Holtby and Allen, the Court of Appeal departed from the traditional binary approach, under which a tort claimant either recovers compensation for his or her entire injury or is altogether denied recovery—depending on whether his or her case against the defendant is more probable than not. Holtby and Allen substituted this approach by (...) the proportionate recovery principle, under which the defendant compensates the claimant for a fraction of his or her injury that represents the defendant's statistical share in that injury. This article analyses this development within the particular domain of indeterminate causation, over which the proportionate recovery principle has been licensed to exercise control. The article claims that this development would constitute an improvement of the law from the perspectives of both optimal deterrence and corrective justice, if the courts properly formulate its scope. First, the proportionate recovery principle needs to be explicitly confined to cases that deal with recurrent wrongs. Second, determination of the defendant's share in the claimant's injury ought to be grounded on the probability of causation, rather than the risk of inflicting that injury. Third, judges must not apply unarticulated intuitions in determining the magnitude of the relevant risk or probability: their decisions in that area would be better informed by the statistical principle of ‘insufficient reason’, also known as the ‘indifference principle’. Fourth, courts are yet to relieve the doctrinal tension between the proportionate recovery principle, as recognized in Holtby and Allen, and the previous rejection of that principle by the House of Lords. Subsequently, the article analyses the House of Lords' decision in Fairchild—yet another instance of indeterminate causation in which proportionate recovery is preferable to the ‘all or nothing’ approach. In this case, the House of Lords allowed the claimants full recovery. This holding was grounded in the Law Lords' innovative approach to indeterminate causation, and the article unfolds the problematics of this approach. Finally, the article offers an adoption of yet another legal mechanism—the ‘evidential damage doctrine’—that replaces liability under uncertainty with liability for uncertainty. (shrink)

Causation is important when considering how an organism maintains health, why disease arises in a healthy person, and how one may intervene to change the course of a disease. This paper explores the form of causative relationships in health, disease and intervention, with particular regard to the pathological and biopsychosocial models. Consistent with the philosophical view of dispositionalism, we believe that objects are the fundamental relata of causation. By accepting the broad scope of the biopsychosocial model, we argue (...) that psychological and social constructs be considered objects. We think that this 'biopsychosocial dispositionalism' offers the flexibility required to describe causation throughout health, disease and intervention pathways. When constructing mechanistic chains to describe causative pathways, we argue that an object will causally connect with others through actions; transfers of energy from one object to another, initiated by the manifestation of one or more dispositional property. Finally, our analysis of causative interactions utilises the concept that a common form of interaction exists between disease and intervention pathways. This common form will always be an object, but the mode of interaction will vary with each disease. We describe how intervention may act through objects being shared between converging mechanistic chains, or through the removal and/or insertion of objects in such chains. We believe that this analysis provides novel insight to the forms of causative transactions that can occur. In addition, we hope that the findings of this analysis represent the first step towards developing a framework for appraising the composition of mechanistic theories. (shrink)

Clarifying the causes of subway construction accidents has an important impact on reducing the probability of accidents and protecting workers’ lives and public property to a greater extent. A total of 138 investigation records of subway construction accidents from 2000 to 2020 were collected in this study. Based on a systemic analysis of 29 well-known accident causation models and the formative process of the subway construction accidents, we extracted the causative factors of subway construction accidents from the collected records. (...) Furthermore, a causation analysis index system of subway accidents was proposed based on fault tree analysis, where we considered subway construction accidents as the top event and the five dimensions, i.e., human, equipment, environment, management, and safety culture, as first-level intermediate events. Moreover, 17 causative factors were considered to be related to the severity of subway construction accidents. It is found that human factors are prone to be critical to high-risk accidents. Finally, a Bayesian network was formed to explore the causative factors of high-risk subway construction accidents. Based on the combined application of FTA and BN, this study discusses the complex influence factors and their action routes to unsafe accidents in subway construction sites, and makes efforts to correspond safety decision basis for the management of China subway construction. (shrink)

In The Biopsychosocial Model of Health and Disease, Derek Bolton and Grant Gillett argue that a defensible updated version of the biopsychosocial model requires a metaphysically adequate account of disease causation that can accommodate biological, psychological, and social factors. This present paper offers a philosophical critique of their account of biopsychosocial causation. I argue that their account relies on claims about the normativity and the semantic content of biological information that are metaphysically contentious. Moreover, I suggest that these (...) claims are unnecessary for a defence of biopsychosocial causation, as the roles of multiple and diverse factors in disease causation can be readily accommodated by a more widely accepted and less metaphysically contentious account of causation. I then raise the more general concern that they are misdiagnosing the problem with the traditional version of the biopsychosocial model. The challenge when developing an explanatorily valuable version of the biopsychosocial model, I argue, is not so much providing an adequate account of biopsychosocial causation, but providing an adequate account of causal selection. Finally, I consider how this problem may be solved to arrive at a more explanatorily valuable and clinically useful version of the biopsychosocial model. (shrink)

Leibniz argued that in natural world, only intra-substantial or immanent causation is possible— the causation that takes place within an individual, when an individual brings about a change in itself. In this dissertation, I address issues arising from Leibniz’s arguments against the rival view that posits a world of causally interacting substances and issues pertaining to Leibniz’s own positive metaphysics of immanent causation and change. -/- Chapter 1 is devoted to stage setting for the remainder of the (...) dissertation. I first offer a historically informed overview of efficient causation and change before introducing Leibniz’s novel views, including his criticisms of competing accounts and his own positive account. After presenting a detailed roadmap of my project, I articulate the idealistic interpretation of Leibniz assumed in this dissertation, where the only genuine substances are simple monads. Finally, I articulate the methodological approaches I employ. -/- In Chapters 2 and 3, I reconstruct and assess Leibniz’s most frequent argument against transeunt causation (the causation that occurs when a substance produces a property in a numerically distinct substance), what I call the “Transference Argument.” Leibniz argued that a created substance’s causing an accident in a numerically distinct substance is possible only if the agent transfers the accident from itself to the patient, where upon transference the agent no longer possesses the accident it transferred. Call the transeunt causal requirement that the agent transfer the accident produced from itself to the patient the “Transference Condition.” -/- Chapter 2 is devoted to two problems with Leibniz’s transference condition. First, Leibniz stated the transference condition throughout his career, but offered little argument for it. Second, God is a transeunt cause in Leibniz’s metaphysics yet God’s causation does not consist in transference. Thus, Leibniz needs a principled way to require transference for creaturely causation while denying that divine transeunt causation consists in transference. Finally, I close off chapter 2 by drawing attention to an important weakness with the transference condition that has not yet been recognized by Leibniz scholars. -/- In Chapter 3, I argue that there is nothing in Leibniz’s ontology that could be transferred from the cause to the recipient of the effect. I first argue that Leibniz’s ontology consists of simple non-corporeal substances and their modifications. Second, I present and articulate a number of important theses Leibniz affirmed about substances and their modifications, which entail that neither could be transferred. I also show that most of these theses were not unique to Leibniz, but were in fact widely endorsed by his predecessors who defended the possibility of creaturely transeunt causation. -/- In chapter 4, I continue the study of the nature of Leibnizian accidents, shifting the focus from their role in Leibniz’s critique of creaturely transeunt causation to their positive role in change and as causal relata, where such accidents are the effects of immanent causation. I shall argue that Leibniz’s thesis that accidents are modifications or limitations allowed him to posit mereologically simple substance that have a multitude of accidents at a time and change accidents over time. -/- In Chapter 5, I address an issue that has divided Leibniz scholars concerning the precise relata in Leibnizian immanent efficient causation. In many passages, Leibniz writes as if it is the substance or individual itself that efficiently causes its later properties or accidents. Call this the “Efficacious-substance” account. The efficacious-substance account is difficult to reconcile with Leibniz’s requirements that change be intelligible and deterministic. In plenty of other passages, he writes as if it is the substances earlier properties or accidents that cause its later accidents. Call this the “Efficacious-accident” account. The efficacious-accident account explains how change is intelligible and deterministic but it faces a “plurality of agents” objection. If a substance’s accidents are the efficient causes of later accidents, then prima facie there is a plurality of efficient causal agents in a substance. This view is incompatible with Leibniz’s requirement that substances be simple, unified entities. -/- Drawing upon a Scholastic distinction made between two kinds of efficient causes— principle quod efficient causes and principle quo efficient causes, I shall argue that for Leibniz, substances are principle quod efficient causes and their appetitions are principle quo efficient causes. This interpretation combines the strengths of the Efficacious-substance and Efficacious-accident accounts while overcoming their weaknesses. There is just one causal agent, the substance, but change is both intelligible and deterministic because as what an agent produces is explained by its appetitions. (shrink)

The fact that causal laws in the social sciences are most realistically expressed as both multivariate and stochastic has a number of very important implications for indirect measurement and generalizability. It becomes difficult to link theoretical definitions of general constructs in a one-to-one relationship to research operations, with the result that there is conceptual slippage in both experimental and nonexperimental research. It is argued that problems of this nature can be approached by developing specific multivariate causal models that incorporate sources (...) of measurement bias, along with the theoretical variables of interest. Many general concepts are defined in such a way that causal assumptions are built into the definitions themselves. Additionally, in any given piece of research it is necessary to omit many variables from consideration, and this is often done without careful consideration of the assumptions required to justify such omissions. Finally, generalization to more inclusive populations or a diversity of settings ordinarily requires one to replace "constants" by variables. It is concluded that the criteria of parsimony, generalizability, and precision are incompatible, given the multivariate nature of social causation, and the author expresses his own preference for sacrificing parsimony in favor of the objectives of achieving increased precision and generalizability of social science laws. (shrink)

Leibniz is almost unique among early modern philosophers in giving final causation a central place in his metaphysical system. All changes in created substances, according to Leibniz, have final causes, that is, occur for the sake of some end. There is, however, no consensus among commentators about the details of Leibniz's views on final causation. The least perfect types of changes that created substances undergo are especially puzzling because those changes seem radically different from paradigmatic (...) instances of final causation. Building on my more general discussion of efficient and final causation in ‘Leibniz on Causation – Part 1,’ I will examine and assess some of the rival interpretations of Leibniz's account of final causation. (shrink)

The current consensus view of causation in physics, as commonly held by scientists and philosophers, has several serious problems. It fails to provide an epistemology for the causal knowledge that it claims physics to possess; it is inapplicable in a prominent area of physics (classical thermodynamics); and it is difficult to reconcile with our everyday use of causal concepts and claims. In this dissertation, I use historical examples and philosophical arguments to show that the interventionist account of causation (...) constitutes a promising alternative for a “physically respectable” account of causation. The interventionist account explicates important parts of the experimental practice of physics and important aspects of the ways in which physical theory is used and applied. Moreover, the interventionist account succeeds where the consensus view of causation in physics fails. I argue that the interventionist account provides an epistemology of causal knowledge in physics that is rooted in experiment. On the interventionist view, there is a close link between experiment and the testing of causal claims. I give several examples of experiments from the early history of thermodynamics that scientists used in interventionist-type arguments. I also argue that interventionist claims made in the context of a physical theory can be epistemically justified by reference to the experimental interventions and observations that serve as evidence for the theory. I then show that the interventionist account of causation is well-suited to the patterns of reasoning that are intrinsic to thermodynamic theory. I argue that interventionist reasoning constitutes the structural foundation of thermodynamic theory, and that thermodynamic theory can provide clear answers to meaningful questions about whether or not a certain variable is a cause of another in a given context. Finally, I argue that the interventionist account offers the prospect of a unification of “physically respectable” causation and our everyday notion of causation. I conclude the dissertation by sketching an anti-foundationalist unification of causation, according to which causal reasoning occurs in the same manner in physics as it does in other branches of life and scientific research. (shrink)

The paper addresses a question concerning George Ellis’s theory of top-down causation by considering a challenge to the “level-picture of nature” which he employs as a foundational element in his theory. According to the level-picture, nature is ordered by distinct and finitely many levels, each characterised by its own types of entities, relations, laws and principles of behavior, and causal relations to their respective neighbouring top- and bottom-level. The branching hierarchy that results from this picture enables Ellis to build (...) his model of modular hierarchical structure for complexity, his account of same-level, bottom-up and top-down causation, of emergence, equivalence-classes and multiple realisability. The three main arguments for the level-picture in Ellis’s works are reconstructed and shown to face serious problems. Finally, the paper presents a possible solution to this challenge by introducing a reformulation of certain fundamental points of Ellis’s theory that does without the level-picture of nature. This allows us to preserve all of his central claims about the model of complexity, the three types of causation, emergence, equivalence-classes and multiple realisability. Any problems pertaining to the level-picture can be remedied in the context of Ellis’s theory of top-down causation. (shrink)

: In this paper, I investigate Leibniz's conception of final causation. I focus especially on the role that Leibnizian final causes play in intentional action, and I argue that for Leibniz, final causes are a species of efficient causation. It is the intentional nature of final causation that distinguishes it from mechanical efficient causation. I conclude by highlighting some of the implications of Leibniz's conception of final causation for his views (...) on human freedom, and on the unconscious activity of substances. (shrink)