In numerous papers Jaegwon Kim argues that nonreductive materialists (i.e., those philosophers who believe that there are no irreducible non-physical objects in the universe, and yet there are irreducible psychological properties which are indispensable in intentional psychological explanations) face two problems. One is that intentional mental properties are not causally relevant; the other is that explanations appealing to these properties are excluded by explanations appealing to physical, in particular, microphysical, properties.1 The first problem can be called the problem of epiphenomenalism. (...) The second problem is what Kim calls the problem of explanatory/causal exclusion. Epiphenomenalism is not just a problem for nonreductive materialists, but a problem for psychology (as a science of psychological explanation), and for anyone who believes that our thought is the cause of our action. Kim argues that the exclusion problem, on the other hand, is especially a problem for nonreductive materialists. Nonreductive materialists typically buy into the principle of the causal closure of the physical domain, according to which "any physical event that has a cause at time t has a physical cause at t..... [I]f we trace the causal ancestry of a physical event, we need never go outside the physical domain." [Kim 1989b, p. 280] One cannot reject this principle unless one is willing to go back to Cartesian dualism. But the problem is, if all causation is done at the physical level, then there is no causal work left for mental kinds. If mental kinds do not play any causal role, then intentional psychological explanations that appeal to mental properties are not warranted. Therefore, Kim concludes, nonreductive materialism is an untenable position. Some nonreductionists (Davidson, Dretske, LePore & Loewer, Jackson & Pettit, for example) have tried to argue for the causal relevancy of mental properties from an.. (shrink)

I introduce "nomic-role nonreductionism" as an alternative to traditional causal-role functionalism in the philosophy of mind. Rather than identify mental properties by a theory that describes their intra-level causal roles via types of inputs, internal states, and outputs, I suggest that one identify mental properties by a more comprehensive theory that also describes inter-level realization roles via types of lower-level engineering, internal mental states, and still higher-level states generated by them. I defend this position on grounds that (...) mental properties should be understood by our best scientific theories, which at present include informatioin about mental engineering. I further defend this claim by a "parity of reasons" argument. Causal-role functionalists are justified to include sensory stimuli in their theory of mind as opposed to, say, the remote causes of sensory stimuli because the former but not the latter are items of direct mental production. But ditto for the system's physical realizations. They too directly produce mental states, only not by "causing" them but by "realizing" them. Engineering realizations and their input triggering conditions work in tandem. In addition, I tell a related but more general metaphysical story about property identity, namely, that the traditional causal theory should be replaced by a more comprehensive nomic theory that individuates properties by their intra-level causal powers as well as their inter-level realization capacities. (shrink)

Jaegwon Kim’s exclusion argument is a general ontological argument, applicable to any properties deemed supervenient on a microproperty basis, including biological properties. It implies that the causal power of any higher-level property must be reducible to the subset of the causal powers of its lower-level properties. Moreover, as Kim’s recent version of the argument indicates, a higher-level property can be causally efficient only to the extent of the efficiency of its micro-basis. In response, I argue that the ontology (...) that aims to capture experimentally based explanations of metabolic control systems and morphogenetic systems must involve causally relevant contextual properties. Such an ontology challenges the exclusiveness of micro-based causal efficiency that grounds Kim’s reductionism, since configurations themselves are inherently causally efficient constituents. I anticipate and respond to the reductionist’s objection that the nonreductionist ontology’s account of causes and inter-level causal relations is incoherent. I also argue that such an ontology is not open to Kim’s overdetermination objection. (shrink)

Political science is divided between methodological individualists, who seek to explain political phenomena by reference to individuals and their interactions, and holists (or nonreductionists), who consider some higher-level social entities or properties such as states, institutions, or cultures ontologically or causally significant. We propose a reconciliation between these two perspectives, building on related work in philosophy. After laying out a taxonomy of different variants of each view, we observe that (i) although political phenomena result from underlying individual attitudes and behavior, (...) individual-level descriptions do not always capture all explanatorily salient properties, and (ii) nonreductionistic explanations are mandated when social regularities are robust to changes in their individual-level realization. We characterize the dividing line between phenomena requiring nonreductionistic explanation and phenomena permitting individualistic explanation and give examples from the study of ethnic conflicts, social-network theory, and international-relations theory. (shrink)

The methodological nonreductionism of contemporary biology opens an interesting discussion on the level of ontology and the philosophy of nature. The theory of emergence (EM), and downward causation (DC) in particular, bring a new set of arguments challenging not only methodological, but also ontological and causal reductionism. This argumentation provides a crucial philosophical foundation for the science/theology dialogue. However, a closer examination shows that proponents of EM do not present a unified and consistent definition of DC. Moreover, they (...) find it difficult to prove that higher-order properties can be causally significant without violating the causal laws that operate at lower physical levels. They also face the problem of circularity and incoherence in their explanation. In our article we show that these problems can be overcome only if DC is understood in terms of formal rather than physical (efficient) causality. This breakdown of causal monism in science opens a way to the retrieval of the fourfold Aristotelian notion of causality. (shrink)

Jaegwon Kim is one of the most preeminent and most influential contributors to the philosophy of mind and metaphysics. This collection of essays presents the core of his work on supervenience and mind with two sets of postscripts especially written for the book. The essays focus on such issues as the nature of causation and events, what dependency relations other than causal relations connect facts and events, the analysis of supervenience, and the mind-body problem. A central problem in the (...) philosophy of mind is the problem of explaining how the mind can causally influence bodily processes. Professor Kim explores this problem in detail, criticizes the nonreductionist solution of it, and offers a modified reductionist solution of his own. Both professional philosophers and their graduate students will find this an invaluable collection. (shrink)

One of the main challenges of the nonreductionist approach to complex structures and phenomena in philosophy of biology is its defense of the plausibility of the theory of emergence and downward causation. The tension between remaining faithful to the rules of physicalism and physical causal closure, while defending the novelty and distinctiveness of emergents from their basal constituents, makes the argumentation of many proponents of emergentism lacking in coherency and precision. In this article I aim at answering the suggestion (...) of several thinkers to redefine emergence and downward causation in terms of the broader Aristotelian view of causation. In addition, I further develop this interdisciplinary conversation to include theological implications of emergentism, analyzed in reference to Aquinas’ understanding of divine action in terms of the same fourfold division of causes—bringing thus natural science, philosophy, and theology into creative and fruitful dialogue. (shrink)

In Part I, the author argued for nonreductive individualism (NRI), an account of the individual-collective relation that is ontologically individualist yet rejects methodological individualism. However, because NRI is ontologically individualist, social entities and properties would seem to be only analytic constructs, and if so, they would seem to be epiphenomenal, since only real things can have causal power. In general, a nonreductionist account is a relatively weak defense of sociological explanation if it cannot provide an account of how social (...) properties can participate in causal relations. In this article, the author extends NRI to address this weakness and provides an account of social causation that he refers to as supervenient causation. Key Words: individualism • collectivism • social realism • social causation. (shrink)

Jaegwon Kim is one of the most pre-eminent and most influential contributors to the philosophy of mind and metaphysics. This collection of essays presents the core of his work on supervenience and mind with two sets of postscripts especially written for the book. The essays focus on such issues as the nature of causation and events, what dependency relations other than causal relations connect facts and events, the analysis of supervenience, and the mind-body problem. A central problem in the (...) philosophy of mind is the problem of explaining how the mind can causally influence bodily processes. Professor Kim explores this problem in detail, criticises the nonreductionist solution of it, and offers a modified reductionist solution of his own. Both professional philosophers and their graduate students will find this an invaluable collection. (shrink)

In this essay, I set out my responses to the following five questions that had been posed: -/- 1. Why were you initially drawn to metaphysics (and what keeps you interested)? 2. What do you consider to be your most important contributions to metaphysics? 3. What do you consider to be the proper method for metaphysics? 4. What do you think is the proper role of metaphysics in relation to other areas of philosophy and other academic disciplines, including the natural (...) sciences? 5. What do you consider to be the most neglected topics in contemporary metaphysics, and what direction would you like metaphysics to take in the future? (shrink)

In this paper we discuss how Causal Decision Theory should be modified to handle a class of problematic cases involving deterministic laws. Causal Decision Theory, as it stands, is problematically biased against your endorsing deterministic propositions (for example it tells you to deny Newtonian physics, regardless of how confident you are of its truth). Our response is that this is not a problem for Causal Decision Theory per se, but arises because of the standard method for assessing (...) the truth of certain counterfactuals. The truth of deterministic laws is `modally fragile' on the standard semantics for counterfactuals: if determinism is true and you were to do otherwise, the laws would be different. We provide two ways of avoiding this problem: 1) supplement the standard semantics for counterfactuals with impossible worlds, or 2) introduce rigid designators into the description of problematic decision situations. We argue that both of these approaches are well-motivated and can be readily incorporated into Lewisian Causal Decision Theory. (shrink)

I take it as given that we manage to linguistically refer to objects we can neither perceive nor uniquely describe. Kripke accounts for this fact by appeal to causal-historical chains of communication. But Evans famously presented what has seemed to many a devastating counterexample to Kripke’s view: the phenomenon of reference-shifting. Here, I’ll agree with critics that Kripke’s view is insufficient to handle cases of reference shift, but I’ll argue for an alternative version of the causal-historical account that (...) is immune to Evans’ counterexample. The key move will be at the foundations; it will require a change in what it is we’re giving a causal-historical account of. Critically, I’ll argue that we should reject two claims associated with the causal-historical picture. First, we should reject the claim that names are used to think of their referents. Second, we should reject the claim that later speakers defer to earlier ones. (shrink)

Modeling mechanisms is central to the biological sciences – for purposes of explanation, prediction, extrapolation, and manipulation. A closer look at the philosophical literature reveals that mechanisms are predominantly modeled in a purely qualitative way. That is, mechanistic models are conceived of as representing how certain entities and activities are spatially and temporally organized so that they bring about the behavior of the mechanism in question. Although this adequately characterizes how mechanisms are represented in biology textbooks, contemporary biological research practice (...) shows the need for quantitative, probabilistic models of mechanisms, too. In this paper we argue that the formal framework of causal graph theory is well-suited to provide us with models of biological mechanisms that incorporate quantitative and probabilistic information. On the ba-sis of an example from contemporary biological practice, namely feedback regulation of fatty acid biosynthesis in Brassica napus, we show that causal graph theoretical models can account for feedback as well as for the multi-level character of mechanisms. However, we do not claim that causal graph theoretical representations of mechanisms are advantageous in all respects and should replace common qualitative models. Rather, we endorse the more balanced view that causal graph theoretical models of mechanisms are useful for some purposes, while being insufficient for others. (shrink)

I argue that Stroud's nonreductionism about meaning is insufficiently motivated. First, given that he rejects the assumption that grasp of an expression's meaning guides or instructs us in its use, he has no reason to accept Kripke's arguments against dispositionalism or related reductive views. Second, his argument that reductive views are impossible because they attempt to explain language “from outside” rests on an equivocation between two senses in which an explanation of language can be from outside language. I offer (...) a partially reductive account of meaning which appeals both to speakers’ dispositions to produce and respond to utterances in naturalistically specifiable ways, and to the normative attitudes they adopt, in so doing, to their own behavior. This account is supported, I argue, by Stroud's early treatment of Wittgenstein's rule-following considerations and in particular of the agreement in primitive judgments or reactions which Wittgenstein takes to be required for linguistic communication. (shrink)

Causal exclusion arguments are taken to threaten the autonomy of the special sciences, and the causal efficacy of mental properties. A recent line of response to these arguments has appealed to “independently plausible” and “well grounded” theories of causation to rebut key premises. In this paper I consider two papers which proceed in this vein and show that they share a common feature: they both require causes to be proportional to their effects. I argue that this feature is (...) a bug, and one that generalises: any attempt to rescue the autonomy of the special sciences, or the efficacy of the mental, from exclusion worries had better not look to proportionality for help. (shrink)

Former discussions of biological generalizations have focused on the question of whether there are universal laws of biology. These discussions typically analyzed generalizations out of their investigative and explanatory contexts and concluded that whatever biological generalizations are, they are not universal laws. The aim of this paper is to explain what biological generalizations are by shifting attention towards the contexts in which they are drawn. I argue that within the context of any particular biological explanation or investigation, biologists employ two (...) types of generations. One type identifies causal regularities exhibited by particular kinds of biological entities. The other type identifies how these entities are distributed in the biological world. (shrink)

This paper expounds that besides the well-known spatio-temporal problem there is a causal problem of entanglement: even when one neglects spatio-temporal constraints, the peculiar statistics of EPR/B experiment is inconsistent with usual principles of causal explanation as stated by the theory of causal Bayes nets. The conflict amounts to a dilemma that either there are uncaused correlations or there are caused independences . I argue that the central ideas of causal explanations can be saved if one (...) accepts the latter horn and explains the unfaithful independences by a stable fine-tuning of the causal parameters. (shrink)

In the literature on scientific explanation two types of pluralism are very common. The first concerns the distinction between explanations of singular facts and explanations of laws: there is a consensus that they have a different structure. The second concerns the distinction between causal explanations and uni.cation explanations: most people agree that both are useful and that their structure is different. In this article we argue for pluralism within the area of causal explanations: we claim that the structure (...) of a causal explanation depends on the causal structure of the relevant fragment of the world and on the interests of the explainer. (shrink)

Much of the recent work on the epistemology of causation has centered on two assumptions, known as the Causal Markov Condition and the Causal Faithfulness Condition. Philosophical discussions of the latter condition have exhibited situations in which it is likely to fail. This paper studies the Causal Faithfulness Condition as a conjunction of weaker conditions. We show that some of the weaker conjuncts can be empirically tested, and hence do not have to be assumed a priori. Our (...) results lead to two methodologically significant observations: (1) some common types of counterexamples to the Faithfulness condition constitute objections only to the empirically testable part of the condition; and (2) some common defenses of the Faithfulness condition do not provide justification or evidence for the testable parts of the condition. It is thus worthwhile to study the possibility of reliable causal inference under weaker Faithfulness conditions. As it turns out, the modification needed to make standard procedures work under a weaker version of the Faithfulness condition also has the practical effect of making them more robust when the standard Faithfulness condition actually holds. This, we argue, is related to the possibility of controlling error probabilities with finite sample size (“uniform consistency”) in causal inference. (shrink)

Jaegwon Kim argues that unreduced mental causes are excluded from efficacy because physical causes are sufficient in themselves. One response to this causal exclusion argument is to embrace some form of overdetermination. In this paper I consider two forms of overdetermination. Independent overdetermination suggests that two individually sufficient causes bring about one effect. This model fails because the sufficiency of one cause renders the other cause unnecessary. Dependent overdetermination suggests that a physical cause is necessary and sufficient for a (...) given effect, but it necessitates a mental cause of the effect as well. This model fails because the necessity of the mental cause renders the physical cause individually insufficient. (shrink)

Agent-causal accounts of free will face two problems. First, such a view needs an account of rational free action, that is, of acting for reasons when one acts freely. And second, an intelligible explication of causation by an agent is required. This paper addresses both of these problems. Free actions are seen as caused both by prior events and by agents. Reasons (or their mental representations) can then be seen as figuring causally when one freely acts for reasons. It (...) is suggested that agent causation can be given a non-reductive theoretical definition, as on some views event causation is. (shrink)

There are two concepts of causes, property causation and token causation. The principle I want to discuss describes an epistemological connection between the two concepts, which I call the Connecting Principle. The rough idea is that if a token event of type Cis followed by a token event of type E, then the support of the hypothesis that the first event token caused the second increases as the strength of the property causal relation of C to E does. I (...) demonstrate the principle, illustrate its application to phylogenies, infections, and rumours, and discuss its consequence for the conceptual distinctness of causal processes from the events they connect. Although I am by no means confident that the Connecting Principle is ultimately correct, it seems to be a useful point of departure into an important aspect of the epistemology of causality. (shrink)

This paper discusses some procedures developed in recent work in machine learning for inferring causal direction from observational data. The role of independence and invariance assumptions is emphasized. Several familiar examples including Hempel’s flagpole problem are explored in the light of these ideas. The framework is then applied to problems having to do with explanatory direction in non-causal explanation.

Research indicates that norms matter for ordinary causal attributions. Across a range of cases in which two agents jointly bring about an outcome, with one violating a norm while the other does not, causal ratings are higher for the agent who violates the norm. Building off such findings, Kominsky et al. note a related phenomenon that they term “causal superseding”—whether or not one agent violates a norm also affects causal ratings for the other agent. Kominsky et (...) al. offer an explanation of this phenomenon and describe the results of four experiments testing their account. In this paper, I explore the proposed phenomenon further. I present a sequence of new studies covering a range of cases, finding that the superseding effect is not as consistent as Kominsky et al. would predict, that the effect does not occur in all of the cases where they say it should, and that it sometimes occurs in cases where they say it shouldn’t. Finally, I offer an alternative deflationary explanation of causal superseding, presenting evidence suggesting that it is simply a context effect. (shrink)

Decision-making typically requires judgments about causal relations: we need to know the causal effects of our actions and the causal relevance of various environmental factors. We investigate how several individuals' causal judgments can be aggregated into collective causal judgments. First, we consider the aggregation of causal judgments via the aggregation of probabilistic judgments, and identify the limitations of this approach. We then explore the possibility of aggregating causal judgments independently of probabilistic ones. Formally, (...) we introduce the problem of causal-network aggregation. Finally, we revisit the aggregation of probabilistic judgments when this is constrained by prior aggregation of qualitative causal judgments. (shrink)

Recent philosophical work has explored the distinction between causal and non-causal forms of explanation. In this literature, topological explanation is viewed as a clear example of the non-causal variety–it is claimed that topology lacks temporal information, which is necessary for causal structure. This paper explores the distinction between topological and causal forms of explanation and argues that this distinction is not as clear cut as the literature suggests. One reason for this is that some explanations (...) involve both topological and causal information. In these “borderline” cases scientists explain some outcome by appealing to the causal topology of the system of interest. These cases help clarify a type of topological explanation that is genuinely causal, but that differs from standard topological and interventionist accounts of explanation. (shrink)

Aristotle’s Metaphysics Θ.8 argument for the priority of actuality to potentiality poses an immediate interpretive problem: the argument uses two distinct tests for priority, one of which threatens to reverse the results of the other. This paper argues that the standard approach to this passage, according to which one thing is prior to another when it satisfies the ontological independence test from Metaphysics Δ.11, fails to secure the argumentative unity of the passage. It introduces a new, causal account of (...) priority which explains both Aristotle’s claims about priority and the way he argues for them. (shrink)

The identity of a natural kind can be construed in terms of its causal profile. This conception is more appropriate to science than two alternatives. The identity of a natural kind is not determined by one causal role because one natural kind can have many causal roles and several functions and because some functions are shared by different kinds. Furthermore, the microstructuralist thesis is wrong: The identity of certain natural kinds is not determined by their microstructure. It (...) is true that if A and B have the same microstructural composition then a sample of a chemical substance A is of the same chemical substance as a sample of B. However, the reverse does not hold. It is not the case that if a sample of a chemical substance A is of the same chemical substance as a sample of B then A and B have the same microstructural composition. This is because a macroscopic NK can be “multiconstituted” by different microstructures. (shrink)

One of Stuart Glennan's most prominent contributions to the new mechanist debate consists in his reductive analysis of higher-level causation in terms of mechanisms (Glennan, 1996). In this paper I employ the causal Bayes net framework to reconstruct his analysis. This allows for specifying general assumptions which have to be satis ed to get Glennan's approach working. I show that once these assumptions are in place, they imply (against the background of the causal Bayes net machinery) that higher-level (...) causation indeed reduces to interactions between component parts of mechanisms. I also briefly discuss the plausibility of these assumptions and some consequences for the mechanism debate. (shrink)

Causal selection has to do with the distinction we make between background conditions and “the” true cause or causes of some outcome of interest. A longstanding consensus in philosophy views causal selection as lacking any objective rationale and as guided, instead, by arbitrary, pragmatic, and non-scientific considerations. I argue against this position in the context of causal selection for disease traits. In this domain, causes are selected on the basis of the type of causal control they (...) exhibit over a disease of interest. My analysis clarifies the principled rationale that guides this selection and how it involves both pragmatic and objective considerations, which have been overlooked in the extant literature. (shrink)

If the laws are deterministic, then standard theories of counterfactuals are forced to reject at least one of the following conditionals: 1) had you chosen differently, there would not have been a violation of the laws of nature; and 2) had you chosen differently, the initial conditions of the universe would not have been different. On the relevant readings—where we hold fixed factors causally independent of your choice—both of these conditionals appear true. And rejecting either one leads to trouble for (...) philosophical theories which rely upon counterfactual conditionals—like, for instance, causal decision theory. Here, I outline a semantics for counterfactual conditionals which allows us to accept both (1) and (2). And I discuss how this semantics deals with objections to causal decision theory from Arif Ahmed. (shrink)

Young children spend a large portion of their time pretending about non-real situations. Why? We answer this question by using the framework of Bayesian causal models to argue that pretending and counterfactual reasoning engage the same component cognitive abilities: disengaging with current reality, making inferences about an alternative representation of reality, and keeping this representation separate from reality. In turn, according to causal models accounts, counterfactual reasoning is a crucial tool that children need to plan for the future (...) and learn about the world. Both planning with causal models and learning about them require the ability to create false premises and generate conclusions from these premises. We argue that pretending allows children to practice these important cognitive skills. We also consider the prevalence of unrealistic scenarios in children's play and explain how they can be useful in learning, despite appearances to the contrary. (shrink)

Social constructionist claims are surprising and interesting when they entail that presumably natural kinds are in fact socially constructed. The claims are interesting because of their theoretical and political importance. Authors like Díaz-León argue that constitutive social construction is more relevant for achieving social justice than causal social construction. This paper challenges this claim. Assuming there are socially salient groups that are discriminated against, the paper presents a dilemma: if there were no constitutively constructed social kinds, the causes of (...) the discrimination of existing social groups would have to be addressed, and understanding causal social construction would be relevant to achieve social justice. On the other hand, not all possible constitutively socially constructed kinds are actual social kinds. If an existing social group is constitutively constructed as a social kind K, the fact that it actually exists as a K has social causes. Again, causal social construction is relevant. The paper argues that (i) for any actual social kind X, if X is constitutively socially constructed as K, then it is also causally socially constructed; and (ii) causal social construction is at least as relevant as constitutive social construction for concerns of social justice. For illustration, I draw upon two phenomena that are presumed to contribute towards the discrimination of women: (i) the poor performance effects of stereotype threat, and (ii) the silencing effects of gendered language use. (shrink)

Lady Mary Shepherd’s critique of Hume’s account of causation, his worries about knowledge of matters of fact, and the contention that it is possible for the course of nature to spontaneously change relies primarily on three premises, two of which – that objects are merely bundles of qualities and that the qualities of an object are individuated by the causal powers contributed by those qualities – anticipate contemporary metaphysical views in ways that she should be getting credit for. The (...) remaining premise – that it is impossible for an object to begin to exist uncaused – seems more old fashioned. I argue that Shepherd can do without her old-fashioned premise and that she provides the materials for arguing that her remaining premises demonstrate a stronger anti-Humeanism than is maintained even by the contemporary representatives of those views, even though she may have to concede more to Humeanism than she would like. (shrink)

Conditional learning, where agents learn a conditional sentence ‘If A, then B,’ is difficult to incorporate into existing Bayesian models of learning. This is because conditional learning is not uniform: in some cases, learning a conditional requires decreasing the probability of the antecedent, while in other cases, the antecedent probability stays constant or increases. I argue that how one learns a conditional depends on the causal structure relating the antecedent and the consequent, leading to a causal model of (...) conditional learning. This model extends traditional Bayesian learning by incorporating causal models into agents’ epistemic states. On this theory, conditional learning proceeds in two steps. First, an agent learns a new causal model with the appropriate relationship between the antecedent and the consequent. Then, the agent narrows down the set of possible worlds to include only those which make the conditional proposition true. This model of learning can incorporate both standard cases of Bayesian learning and the non-uniform learning required to learn conditional information. (shrink)

This article defends the use of interventionist counterfactuals to elucidate causal and explanatory claims against criticisms advanced by James Bogen and Peter Machamer. Against Bogen, I argue that counterfactual claims concerning what would happen under interventions are meaningful and have determinate truth values, even in a deterministic world. I also argue, against both Machamer and Bogen, that we need to appeal to counterfactuals to capture the notions like causal relevance and causal mechanism. Contrary to what both authors (...) suppose, counterfactuals are not "unscientific" - a substantial tradition within statistics and the causal modelling literature makes heavy use of them. (shrink)

This article compares causal and constitutive explanation. While scientific inquiry usually addresses both causal and constitutive questions, making the distinction is crucial for a detailed understanding of scientific questions and their interrelations. These explanations have different kinds of explananda and they track different sorts of dependencies. Constitutive explanations do not address events or behaviors, but causal capacities. While there are some interesting relations between building and causal manipulation, causation and constitution are not to be confused. Constitution (...) is a synchronous and asymmetric relation between relata that cannot be conceived as independent existences. However, despite their metaphysical differences, the same key ideas about explanation largely apply to both. Causal and constitutive explanations face similar challenges (such as the problems of relevance and explanatory regress) and both are in the business of mapping networks of counterfactual dependence—i.e. mechanisms—although the relevant counterfactuals are of a different sort. In the final section the issue of developmental explanation is discussed. It is argued that developmental explanations deserve their own place in taxonomy of explanations, although ultimately developmental dependencies can be analyzed as combinations of causal and constitutive dependencies. Hence, causal and constitutive explanation are distinct, but not always completely separate forms of explanation. (shrink)

What does it mean to say that mind-body dualism is causally problematic in a way that other mind-body theories, such as the psychophysical type identity theory, are not? After considering and rejecting various proposals, I advance my own, which focuses on what grounds the causal closure of the physical realm. A metametaphysical implication of my proposal is that philosophers working without the notion of grounding in their toolkit are metaphysically impoverished. They cannot do justice to the thought, encountered in (...) every introductory class in the philosophy of mind, that dualism has a special problem accounting for mental causation. (shrink)

ABSTRACT Causes must be distinct from their effects. If the temperature in a room is 15°F, this can cause water pipes to freeze. However, the temperature’s being 15°F is not a cause of the temperature’s being below the freezing point. In general, conceptual, logical, mathematical, and other non-causal dependence relations should not be misclassified as causal. In this paper, I discuss how interventionist theories of causation can meet the challenge of distinguishing between (direct or indirect) causal relations (...) and dependence relations that are due to non-causal factors. I survey several approaches to solving this problem, and propose a novel criterion based on the notion of supervenience. According to this criterion, variables are sufficiently distinct to qualify as the relata of a causal relation iff they do not have overlapping minimal supervenience bases. (shrink)

Causal closure arguments against interactionist dualism are currently popular amongst physicalists. Such an argument appeals to some principles of the causal closure of the physical, together with certain other premises, to conclude that at least some mental events are identical with physical events. However, it is crucial to the success of any such argument that the physical causal closure principle to which it appeals is neither too strong nor too weak by certain standards. In this paper, it (...) is argued that various forms of naturalistic dualism, of an emergentist character, are consistent with the strongest physical causal closure principles that can plausibly be advocated. (shrink)

Griffiths et al. (2015) have proposed a quantitative measure of causal specificity and used it to assess various attempts to single out genetic causes as being causally more specific than other cellular mechanisms, for example, alternative splicing. Focusing in particular on developmental processes, they have identified a number of important challenges for this project. In this discussion note, I would like to show how these challenges can be met.

Causal decision theory (CDT) cares only about the effects of a contemplated act, not its causes. The article constructs a case in which CDT consequently recommends a bet that the agent is certain to lose, rather than a bet that she is certain to win. CDT is plainly giving wrong advice in this case. It therefore stands refuted. 1 The Argument2 The Argument in More Detail2.1 The betting mechanism2.2 Soft determinism2.3 The content of P 2.4 The argument again3 The (...) Descriptive Premise3.1 Causal decision theory3.2 Causal decision theory prefers A14 The Normative Premise5 Objections5.1 Table 1 and Table 2 are misleading5.2 The agency theory of causation5.3 The payment mechanism5.4 Newcomb’s problem5.5 Against the normative premise5.6 Drop soft determinism. (shrink)

Horgan (1993) proposed that "superdupervenience" - supervenience preserving physicalistic acceptability - is a matter of robust explanation. I argued against him (1999) that (as nearly all physicalist and emergentist accounts reflect) superdupervenience is a matter of Condition on Causal Powers (CCP): every causal power bestowed by the supervenient property is identical with a causal power bestowed by its base property. Here I show that CCP is, as it stands, unsatisfactory,for on the usual understandings of causal power (...) bestowal, it is trivially satisfied or falsified. I offer a revision of CCP which incorporates the evident fact that causal powers are grounded in fundamental forces. (shrink)