Abstract

Intrauterine fetal hypoxia occupies one of the first places in the structure of the causes of perinatal diseases and mortality. This pathology, as a rule, is a consequence of placental insufficiency, which accompanies almost all complications of pregnancy - miscarriage, gestosis, developmental delay or fetal death, premature birth, acute or chronic infection [2,3,10]. Recent epidemiological studies indicate the leading role of brain lesions that occurred during the perinatal period in the further dysadaptation, and in some cases, disability in children. Thus, in the structure of children's disability, lesions of the nervous system account for about 50% [1,5,11]. Thus, 35-40% of disabled children are disabled due to perinatal lesions of the nervous system [4,6]. Therefore, the issues of hypoxic-ischemic encephalopathy remain relevant.