The fact that causal laws in the social sciences are most realistically expressed as both multivariate and stochastic has a number of very important implications for indirect measurement and generalizability. It becomes difficult to link theoretical definitions of general constructs in a one-to-one relationship to research operations, with the result that there is conceptual slippage in both experimental and nonexperimental research. It is argued that problems of this nature can be approached by developing specific multivariate causal models that incorporate sources (...) of measurement bias, along with the theoretical variables of interest. Many general concepts are defined in such a way that causal assumptions are built into the definitions themselves. Additionally, in any given piece of research it is necessary to omit many variables from consideration, and this is often done without careful consideration of the assumptions required to justify such omissions. Finally, generalization to more inclusive populations or a diversity of settings ordinarily requires one to replace "constants" by variables. It is concluded that the criteria of parsimony, generalizability, and precision are incompatible, given the multivariate nature of social causation, and the author expresses his own preference for sacrificing parsimony in favor of the objectives of achieving increased precision and generalizability of social science laws. (shrink)

The fact that causal laws in the social sciences are most realistically expressed as both multivariate and stochastic has a number of very important implications for indirect measurement and generalizability. It becomes difficult to link theoretical definitions of general constructs in a one-to-one relationship to research operations, with the result that there is conceptual slippage in both experimental and nonexperimental research. It is argued that problems of this nature can be approached by developing specific multivariate causal models that incorporate sources (...) of measurement bias, along with the theoretical variables of interest. Many general concepts are defined in such a way that causal assumptions are built into the definitions themselves. Additionally, in any given piece of research it is necessary to omit many variables from consideration, and this is often done without careful consideration of the assumptions required to justify such omissions. Finally, generalization to more inclusive populations or a diversity of settings ordinarily requires one to replace constants by variables. It is concluded that the criteria of parsimony, generalizability, and precision are incompatible, given the multivariate nature of social causation, and the author expresses his own preference for sacrificing parsimony in favor of the objectives of achieving increased precision and generalizability of social science laws. (shrink)

It is argued in this paper that although much attention has been paid to causal chains and common causes within the literature on probabilistic causality, a primary virtue of that approach is its ability to deal with cases of multiple causation. In doing so some ways are indicated in which contemporary sine qua non analyses of causation are too narrow (and ways in which probabilistic causality is not) and an argument by Reichenbach designed to provide a basis (...) for the asymmetry of causation is refined. The importance of referring causal claims to an abstract model is also emphasized. (shrink)

The paper argues that mental causation can be explained from the sufficiency of counterfactual dependence for causation together with relatively weak assumptions about the metaphysics of mind. If a physical event counterfactually depends on an earlier physical event, it also counterfactually depends on, and hence is caused by, a mental event that correlates with (or supervenes on) this earlier physical event, provided that this correlation (or supervenience) is sufficiently modally robust. This account of mental causation is consistent (...) with the overdetermination of physical events by mental events and other physical events, but does not entail it. (shrink)

Inhaltsverzeichnis/Table of Contents: Introduction. Marc SLORS: Epiphenomenalism and Cross-Realization Induction. Michael PAUEN: Is Type Identity Incompatible with Multiple Realization? Sven WALTER: Need Multiple Realizability Deter the Identity-Theorist? Achim STEPHAN: Emergentism, Irreducibility, and Downward Causation. Carl GILLETT: The Varieties of Emergence: Their Purposes, Obligations and Importance. Wim DE MUIJNCK: Causation by Relational Properties. Albert NEWEN & Rimas ČUPLINSKAS: Mental Causation: A Real Phenomenon in a Physicalistic World without Epiphenomenalism or Overdetermination. Bernd LUDWIG: Warum kommen „mentale Ursachen“ (...) physikalischen Erklärungen eigentlich nicht in die Quere? Einige grundsätzliche Überlegungen zur Verwendung des Ausdrucks „A verursacht B“ im Umkreis moderner naturwissenschaftlicher Theorien. Besprechungen/Review Articles. Buchnotizen/Critical Notes. (shrink)

In this paper, we analyse the fruitfulness of Ronald Giere’s comparative model for causation in populations. While the original model was primarily developed to capture the meaning of causal claims in the biomedical and health sciences, we want to show that the model is not only useful in these domains, but can also fruitfully be applied to other scientific domains. Specifically, we demonstrate that the model is fruitful for characterizing the meaning of causal claims found in classical genetics, epidemiology (...) and electoral sociology. Additionally, we propose an adapted comparative model which is needed to get a grip on higher level causal claims. We show that such claims are present in population genetics, epidemiology and electoral systems research. We conclude with some reflections on the implications of our findings for the issue of causal pluralism. (shrink)

In many people, caffeine causes slight muscle tremors, particularly in their hands. In general, the Caffeine → Muscle Tremors causal connection is a noisy one: someone can drink coffee and experience no hand shaking, and there are many other factors that can lead to muscle tremors. Now suppose that Jane drinks several cups of coffee and then notices that her hands are trembling; an obvious question is: did this instance of coffee drinking cause this instance of hand-trembling? Structurally similar questions (...) arise throughout everyday life: Did this pressing of the ‘k’ key cause this change in the pixels on the computer monitor? Did these episodes of smoking cause this lung cancer? Did this studying cause this test score? And so on. These questions all ask about singular causation in a particular situation, in contrast with general causation across multiple cases. They are thus particularly salient in situations in which we care about that specific case, as in many legal contexts, social interactions, physical explanations of anomalous events, and more. (shrink)

The aim of this paper is to defend the causal homogeneity of functional, mental properties against Kim’s attack. It is argued that (a) token identity is sufficient for mental causation, that (b) token identity implies a sort of functional reduction, but that (c) nonetheless functional, mental properties can be causally homogeneous despite being multiply realizable: multiple composition is sufficient for multiple realizability, but multiple composition does not prevent the realizers from having their pertinent effects in common. (...) Thus, the causal exclusion problem provides no argument for abandoning the position that there are functional, mental properties that are natural kind properties. (shrink)

The idea of agent causation—that a system such as a living organism can be a cause of things in the world—is often seen as mysterious and deemed to be at odds with the physicalist thesis that is now commonly embraced in science and philosophy. Instead, the causal power of organisms is attributed to mechanistic components within the system or derived from the causal activity at the lowest level of physical description. In either case, the ‘agent’ itself (i.e., the system (...) as a whole) is left out of the picture entirely, and agent causation is explained away. We argue that this is not the right way to think about causation in biology or in systems more generally. We present a framework of eight criteria that we argue, collectively, describe a system that overcomes the challenges concerning agent causality in an entirely naturalistic and non-mysterious way. They are: (1) thermodynamic autonomy, (2) persistence, (3) endogenous activity, (4) holistic integration, (5) low-level indeterminacy, (6) multiple realisability, (7) historicity, (8) agent-level normativity. Each criterion is taken to be dimensional rather than categorical, and thus we conclude with a short discussion on how researchers working on quantifying agency may use this multidimensional framework to situate and guide their research. (shrink)

I survey recent work on mental causation. The discussion is conducted under the twin presumptions that mental states, including especially what subjects believe and desire, causally explain what subjects do, and that the physical sciences can in principle give a complete explanation for each and every bodily movement. I start with sceptical discussions of various views that hold that, in some strong sense, the causal explanations offered by psychology are autonomous with respect to those offered by the physical sciences. (...) I then proceed to views that see the problem of mental causation as that of identifying where in the physical story about us and our world lie the parts that in effect tell us abut mental causation - the kind of position that is pretty much standard in the cognitive science community - and consider issues raised by various forms of functionalism and externalism. The general thrust of my discussion is sympathetic to the story about mental causation suggested by those type-type versions of the mind-brain identity theory that allow for the possiblity of multiple realisability. I include a brief discussion of how a map-system account of belief, by contrast with a language of thought one, should understand explanations of behaviour in terms of what a subject believes. (shrink)

This chapter contains section titled: The World's Existence as an Item of Evidence Evidence from the World's Causal Orderliness Evidence from Cosmic Fine‐Tuning Philosophical Attempts to Shrug Off the Fine‐Tuning Multiple Universes and Observational Selection What Observational Selection Maybe Couldn't Make Unmysterious Observational Selection in Pantheism's Cosmos Conclusions Postscript.

Despite the fact that the nature of the properties of causation is rarely discussed within the mental causation debate, the implicit assumption is that they are universals as opposed to tropes. However, in recent literature on the problem of mental causation, a new solution has emerged which aims to address the problem by appealing to tropes. It is argued that if the properties of causation are tropes rather than universals, then a psychophysical reductionism can be advanced (...) which does not face the problem of multiple realizability. However, the 'trope solution' rests upon the assumption that one can combine a trope monism with a type dualism. I argue that such a combination cannot be allowed. Given a plausible interpretation of types within a trope ontology, trope monism in fact entails type monism. Consequently, if one identifies mental tropes with physical tropes, one must also identify mental and physical types and in doing so face a modified version of the multiple realizability argument. (shrink)

In The Biopsychosocial Model of Health and Disease, Derek Bolton and Grant Gillett argue that a defensible updated version of the biopsychosocial model requires a metaphysically adequate account of disease causation that can accommodate biological, psychological, and social factors. This present paper offers a philosophical critique of their account of biopsychosocial causation. I argue that their account relies on claims about the normativity and the semantic content of biological information that are metaphysically contentious. Moreover, I suggest that these (...) claims are unnecessary for a defence of biopsychosocial causation, as the roles of multiple and diverse factors in disease causation can be readily accommodated by a more widely accepted and less metaphysically contentious account of causation. I then raise the more general concern that they are misdiagnosing the problem with the traditional version of the biopsychosocial model. The challenge when developing an explanatorily valuable version of the biopsychosocial model, I argue, is not so much providing an adequate account of biopsychosocial causation, but providing an adequate account of causal selection. Finally, I consider how this problem may be solved to arrive at a more explanatorily valuable and clinically useful version of the biopsychosocial model. (shrink)

In "Causes and "If P, Even If X, still Q," Philosophy 57 (July 1982), Ted Honderich cites my "The Direction of Causation and the Direction of Conditionship," journal of Philosophy 73 (April 22, 1976) as an example of an account of causal priority that lacks the proper character. After emending Honderich's description of the proper character, I argue that my attempt to account for one-way causation in terms of one-way causal conditionship does not totally lack it. Rather than (...) emphasize the singularity of an effect, as Honderich does, I emphasize the multiplicity of independent factors in a causal circumstance. (shrink)

The paper addresses a question concerning George Ellis’s theory of top-down causation by considering a challenge to the “level-picture of nature” which he employs as a foundational element in his theory. According to the level-picture, nature is ordered by distinct and finitely many levels, each characterised by its own types of entities, relations, laws and principles of behavior, and causal relations to their respective neighbouring top- and bottom-level. The branching hierarchy that results from this picture enables Ellis to build (...) his model of modular hierarchical structure for complexity, his account of same-level, bottom-up and top-down causation, of emergence, equivalence-classes and multiple realisability. The three main arguments for the level-picture in Ellis’s works are reconstructed and shown to face serious problems. Finally, the paper presents a possible solution to this challenge by introducing a reformulation of certain fundamental points of Ellis’s theory that does without the level-picture of nature. This allows us to preserve all of his central claims about the model of complexity, the three types of causation, emergence, equivalence-classes and multiple realisability. Any problems pertaining to the level-picture can be remedied in the context of Ellis’s theory of top-down causation. (shrink)

While it is generally agreed that the concept of homology refers to individuated traits that have been inherited from common ancestry, we still lack an adequate account of trait individuation or inheritance. Here I propose that we utilize a counterfactual criterion of causation to link each trait with a developmental-causal (DC) gene. A DC gene is made up of the genetic information (which might or might not be physically contiguous in the genome) that is needed for the production of (...) the organismic attributes that comprise the trait. I argue that individuated traits—phenes—correspond to organismic features that are caused by DC genes. Using such an approach, we can define a DC map, which shows the relations between each pair of phenes and provides a succinct summary of genotype-phenotype relationships and phenotypic complexity. Phenes in parents and offspring are judged to be homologous if their DC genes are composed of orthologous genetic factors. When comparing more distantly related organisms, traits are homologous when linked by a chain of parent-offspring homologs along the path of ancestry that links the two organisms. There are three possible ways to deal with the potential for multiple equivalent DC genes: maximal, minimal, and consensus homology. Whereas maximal homology has limited utility, the other two approaches have value and can help to guide research at the intersection of evolution and development. (shrink)

I propose an analysis of harm in terms of causation: harm is when a subject is caused to be worse off. The pay-off from this lies in the details. In particular, importing influential recent work from the causation literature yields a contrastive-counterfactual account. This enables us to incorporate harm's multiple senses into a unified scheme, and to provide that scheme with theoretical ballast. It also enables us to respond effectively to previous criticisms of counterfactual accounts, as well (...) as to sharpen criticisms of rival views. (shrink)

Although Ross & Spurrett (R&S) successfully fend off the threat of Kim's “supervenience argument” by showing that it conflates different notions of causation, their proposal for a dynamic systems answer to the mind-body problem is itself yet another supervenience claim in need of an explanation that justifies it. The same goes for their notion of “multiple supervenience.”.

This article critiques R. Keith Sawyer’s theory of social causation from his 2005 book Social Emergence. It considers his use of analogy with the philosophy of mind, his account of individual agency, the legacy of Emile Durkheim, the concepts of supervenience, multiple realization, and wild disjunction, and the role of history in social causation. Sawyer’s theory is also evaluated in terms of two examples of empirical research: his own micro-sociological studies into group creativity; and Margaret Archer’s macro-sociology (...) of education systems. (shrink)

In his latest book Physicalism, or Something near Enough, Jaegwon Kim argues that his version of functional reductionism is the most promising way for saving mental causation. I argue, on the other hand, that there is an internal tension in his position: Functional reductionism does not save mental causation if Kim’s own supervenience argument is sound. My line of reasoning has the following steps: (1) I discuss the supervenience argument and I explain how it motivates Kim’s functional reductionism; (...) (2) I present what I call immense multiple realization, which says that macro-properties are immensely multiply realized in determinate micro-based properties; (3) on that background I argue that functional reductionism leads to a specified kind of irrealism for mental properties. Assuming that such irrealism is part of Kim’s view, which Kim himself seems to acknowledge, I argue that Kim’s position gets the counterfactual dependencies between macro-causal relata wrong. Consequently, his position does not give a conservative account of mental causation. I end the paper by discussing some alternative moves that Kim seems to find viable in his latest book. I argue on the assumption that the supervenience argument is sound, so the discussion provides further reasons to critically reevaluate that argument because it generalizes in deeply problematic ways. (shrink)

Multiple supervenience is a problematic notion whose role can well be served by a contextualized or properly restricted standard notion of supervenience. It is furthermore not needed to defend functionalism against Kim's charge that cross-classifying taxonomies imply a serious form of dualism; nor does Ross & Spurrett's (R&S's) Kitcherian account of the metaphysics of causation crucially depend on multiple supervenience.

In this dissertation, I try to answer the following somewhat Kantian question: How is mental causation possible? For the discussion of the nature of mental properties, I accept the two tenets of functionalism: the multiple realizability thesis and the functional conception of mental properties as causal intermediaries between physical causes and effects. I then introduce the following two principles: the principle of physical realization and the principle of causal power identity. It is the principle of physical realization that (...) dictates that every mental property be physically realized for its instantiation. The principle of causal power identity ensures that the causal power of a mental property is identical with those of its physical realizers. I suggest that the realization relation between the mental and the physical be understood in such a way that a mental property has causal powers identical with those of its physical realizers, but that the mental property itself is not reduced to its physical realizers because the two kinds of properties are not identical. Mental properties are realized in physical properties, and the causal powers of mental properties are identical with those of physical properties; therefore, mental properties are as causally efficacious as their physical realizers. Since mental properties are not reducible to physical properties, they enjoy autonomy with the full causal powers of their realizers. To conclude, mental properties do not lose out to epiphenomenalism; and they have their own autonomy. (shrink)

Causation is one of the most controversial topics in philosophy. There is a wide range of philosophical accounts of causation, for example, the regularity account, the probabilistic account, the counterfactual account, the interventionist account, which can be all classified as ‘difference-making’ accounts; and the mechanistic account. Many argue that only one of these accounts is correct as there is only one type of causal relation (causal monism), while others maintain that there are multiple types of causation (...) (causal pluralism). In addition, there are eliminativists argue that science has no need of causation at all, while primitivists maintain that causation is unanalysable. Recently, the difference-making and mechanistic approaches have dominated recent philosophical discussion of causation. Other approaches and positions have been insufficiently discussed and assessed, especially in the context of philosophy of science. This volume explores and examines alternative approaches to causation. It revisits causal primitivism and causal eliminativism in the context of recent literature. It further explores the pluralistic approach, the fictionalist approach, the inferentialist approach, and the informational approach. It also examines the application of the dispositional approach, the epistemic approach, and the powerful particulars approach to the natural and social sciences. Overall, the volume is complementary to the recent discussion on the difference-making and mechanistic approaches and sheds new light on the metaphysical, epistemological, conceptual and methodological issues on causation. As such, it provides foundations for further research and teaching of this hotly debated topic. (shrink)

A critical analysis of recent interventionist responses to the causal exclusion problem is presented. It is argued that the response can indeed offer a solution to the problem, but one that is based on renouncing the multiple realizability thesis. The account amounts to the rejection of nonreductive physicalism and would thus be unacceptable to many. It is further shown that if the multiple realizability thesis is brought back in and conjoined with the interventionist notion of causation, inter-level (...) causation is ruled out altogether. (shrink)

This essay, written by a biologist, a microbial ecologist, a biological anthropologist, and an anthropologist‐historian, examines tensions and translations in microbiome research on animals in the laboratory and field. The authors trace how research questions and findings in the laboratory are extrapolated into the field and vice versa, and the shifting evidentiary standards that these research settings require. Showing how complexities of microbiomes challenge traditional standards of causation, the authors contend that these challenges require new approaches to inferences used (...) in ecology, anthropology, and history. As social scientists incorporate investigations of microbial life into their human studies, microbiome researchers venture into field settings to develop mechanistic understandings about the functions of complex microbial communities. These efforts generate new possibilities for cross‐fertilizations and inference frameworks to interpret microbiome findings. Microbiome research should integrate multiple scales, levels of variability, and other disciplinary approaches to tackle questions spanning conditions from the laboratory to the field. (shrink)

Thomas Szasz''s emphasis on goal-oriented behavior and moral responsibility has raised profound theoretical questions about an ancient and enduring problem in philosophy, the relationships amongfree will, determinism, and moral responsibility. Two early thinkers, Jonathan Edwards and Aristotle, have both contributed to an understanding of this dilemma. Edwards (1754) demonstrated that the concept of man as a moral agent and the doctrine of philosophical necessity are inextricably intertwined, in opposition to the tenets of contingency, moral indifference, and self-determining volition. However, his (...) argument rested on efficient causationalone. For further light on the problem we can turn to Aristotle''s writings, notably the distinguishing criteria of the practical sciences (including psychology) as opposed to the theoretical and the productive sciences; and multiple causation, one of the most powerful tools for intellectual analysis ever invented, which includes material, formal, and final causes as well as the efficient cause. The implications of Szasz''s work force a re-examination of the contributions of both Edwards and Aristotle and make them relevant to contemporary psychiatry. (shrink)

The paper discusses the evolving concept of causationin epidemiology and its potential interaction with logic and scientific philosophy. Causes arecontingent but the necessity which binds them totheir effects relies on contrary-to-fact conditionals,i.e. conditional statements whose antecedent is false.Chance instead of determinism plays a growing role inscience and, although rarely acknowledged yet, inepidemiology: causes are multiple and chancy; a priorevent causes a subsequent event if the probabilitydistribution of the subsequent event changesconditionally upon the probability of the prior event.There are no (...) known sufficient causes in epidemiology.We merely observe tendencies toward sufficiency ortendencies toward necessity: cohort studies evaluatethe first tendencies, and case-control studies thelatter.In applied sciences, such as medicine andepidemiology, causes are intrinsically connected withgoals and effective strategies: they are recipewhich have a potential harmful or sucessful use; theyare contrastive since they make a differencebetween circumstances in which they are present andthose in which they are absent: causes do notexplain Event E but event E rather thaneven F. Causation is intrinsically linked with thenotion of ``what is pathological''.Any definition of causation will inevitably collapseinto the use made of epidemiologic methods. Theprogressive methodological sophistication of the lastforty years is in perfect alignment with a gradualimplicit overhaul of our concept of causation. (shrink)

Some recent arguments defending the genuine causal efficacy of the mental have been relying on empirical research on neuroprosthetics. This essay presents a critical analysis of these arguments. The problem of mental causation, and the basic idea and results of neuroprosthetics are reviewed. It is shown how appealing to the research on neuroprosthetics can be interpreted to give support to the idea of mental causation. However, it does so only in a rather deflationary sense: by holding the mental (...) identical with the neural. So contrary to what the arguments have been assuming, neuroprosthetics cannot be used to argue for nonreductive physicalism. It can rather be taken to illustrate just the opposite: how the mental and the physical are identical. (shrink)

Between 1653 and 1655 Margaret Cavendish makes a radical transition in her theory of matter, rejecting her earlier atomism in favour of an infinitely-extended and infinitely-divisible material plenum, with matter being ubiquitously self-moving, sensing, and rational. It is unclear, however, if Cavendish can actually dispense of atomism. One of her arguments against atomism, for example, depends upon the created world being harmonious and orderly, a premise Cavendish herself repeatedly undermines by noting nature’s many disorders. I argue that her supposed difficulties (...) with atomism expose a deeper tension in her work between two fundamental metaphysical commitments each of which has substantial philosophical support: her monist theory of the material world (which maintains that there exists just one natural substance which is the single principal cause) and her occasional theory of causation (which requires multiple finite principal causes in nature -- causes that might be considered individual substances). Her monism undermines atomism while her theory of occasional cause seems to rest on a conception of nature that would be especially friendly to atomism. I argue further that we can solve this tension within a Cavendishian framework in such a way as to preserve her theory of causation and her monism, but that this solution depends upon our taking her monism in a particular (and weak) form. I finally note that we can best make sense of her unique and interesting form of monism by acknowledging her social-political motivations in addition to her motivations in natural philosophy. (shrink)

Allow me to recapitulate some territory that will be familiar to most readers. Here is how the problem of mental causation has typically been set up since shortly after the onset of non-reductive physicalism. It is now widely assumed that the realm of the physical is causally closed: every physical event has a complete physical cause, a cause that is sufficient for the event’s occurrence. This apparently leaves us with a limited number of options concerning psychological causation, none (...) of which appear hugely attractive. Either: (a) the psychological is epiphenomenal and can have no causal impact on the physical, or (b) the psychological is identical with the physical, or (c) thoughts and actions are all over-determined, each one having two distinct sufficient causes. Option (b) subdivides into two further options. Either (b1) the psychological reduces to the physical and every psychological property is identical with some physical property, or (b2) token psychological events are identical with or constituted from token physical events but psychological properties are not identical with physical properties. (b1) is widely held to be inconsistent with the multiple realisation of the psychological by the physical. And (b2) appears to bring us back to the original problematic, with the properties as the locus of tension. If one event causes another it does so in virtue some of its properties and not others. If I throw a stone at a window and the window breaks, it is because the stone was hard and heavy that it broke the window and not, say, because it was grey and millions of years old. The properties in virtue of which an event has a particular effect are typically called the ‘causally efficacious properties of the cause with respect to the effect.’ Suppose, then that token neural event causes an action. We can ask ‘Does it do so in virtue of its physical properties or its psychological properties?’ and we are back to choosing between options (a) and (c) or returning to (b1).. (shrink)

A great deal has been written over the past decade defending ‘higher-level’ causes by arguing that overdetermination is more complex than many philosophers initially thought. Although two shooters overdetermine the death of a firing squad victim, a baseball and its parts do not overdetermine the breaking of a window. But while these analyses of overdetermination have no doubt been fruitful, the focus on overdetermination—while ignoring other varieties of causal relation—has limited the discussion. Many of the cases of interest resemble joint (...) causes or a cause necessitating a simultaneous epiphenomenon as much as they resemble overdeterminers. If we are to fully understand higher-level causation, we need to distinguish it from these causal relations as well. This paper is dedicated to the task, focusing especially on the ‘threat’ that higher-level causes are epiphenomena necessitated by lower-level causes. (shrink)

This paper tries to remove some obstacles standing in the way of considering mental properties as both genuine natural kinds and causally efficacious rather than epiphenomena. As the case of temperature shows, it is not justified to conclude from a property being multiply realizable to it being irreducible. Yet Kim's argument to the effect that if a property is multiply realizable with a heterogeneous reduction base then it cannot be a natural kind and possesses only derivative “epiphenomenal” causal efficacy is (...) not conclusive either. The fact that temperature is, but jade is not, a natural kind cannot be established by comparing the heterogeneity of their respective reduction bases, but rather by the fact that the former is and the latter is not embedded in laws of nature. (shrink)

Discrimination law is a possible application of the methods of causal modelling. With it, it brings the possibility of direct statistical evidence on counterfactual questions, something that traditional techniques like multiple regression lack. The kinds of evidence that causal modelling can provide, in large part due to its attention to counterfactuals, is very close to the key question that we ask of jurors in discrimination cases. With this new kind of evidence comes new opportunities. We can better proportion punitive (...) damages to the severity of the discrimination that manifests in a hiring process. We can avoid making certain kinds of assumptions regarding the relationship between protected classes and hiring qualifications that other statistical methods demand from statisticians. We can also distribute restitution to individual claimants in a way that is proportionate to how their application was treated in the hiring process. Here we explore where and how causal modelling can be useful in discrimination law and policy. What elements of law provide friction with this mode of gathering statistical evidence, what new possibilities does it reveal, and how does this integrate with prior judgments regarding statistical evidence? (shrink)

The thesis of multiple realisation that Borsboom et al. are relying on should not be taken for granted. In dissolving the apparent multiple realisation, the reductionist research strategies in psychopathology research (the Research Domain Criteria [RDoC] framework, in particular) are bound to lead to eliminativism rather than reductionism. Therefore, Borsboom et al. seem to be aiming at a wrong target.

In this paper I present a metaphysically minimalist but theoretically strong version of fact causation, in which the causal relata constitute a full Boolean algebra, mirroring the entailment relation of the sentences that express them. I suggest a generalization of the notion of multiple realizability of causes in terms of specificity of facts, and employ this in an interpretation of what goes on in cases of apparently redundant causation.

The paper attempts to reconcile two very different approaches to the concept of causation. In the original form, it is the opposition found in Laplace between his doctrine of constant and variable causes on the one hand and his mechanistic determinism on the other. This tension was described clearly only by Maxwell who stressed the contrast between the statistical and the dynamical method (calling the latter also the historical or strictly kinetic method). A similar dichotomy surfaces in the work (...) of Wesley Salmon who distinguished statistical from aleatory causation. Acknowledging that we seem to have conflicting intuitions about causality and explanation, I argue that assigning probabilistic laws a fundamental role in scientific explanation does not conflict with the assumption of Laplacian determinism. This is due to the fact that in most systems of interest, we confront a complex interaction of multiple causal factors and an extreme sensitivity to small variations of initial conditions. (shrink)

In lieu of an abstract, here is a brief excerpt of the content:Psychophysical Causation and a Pragmatist Approach to Human BehaviorDavid H. Brendel (bio)Keywordsmind-body problem, philosophy, pragmatism, psychology, psychophysical causationJochen Fahrenberg and Marcus Cheetham have performed a valuable service by conducting and presenting an empirical study of some basic philosophical assumptions of psychologists, philosophers, and scientists. Well-designed, large-scale empirical studies of this kind are all too rare in the literature. Those of us interested in the human sciences are rather (...) in the dark about the assumptions of others working in our own and related fields. For the most part, we are privy only to the philosophical assumptions of friends and colleagues we know intimately and of those relative few who publish their views. We can only speculate about the views of the countless others with whom we have not had the opportunity to establish direct philosophical discourse. The findings presented by Fahrenberg and Cheetham give us an intriguing glimpse at some of the key philosophical assumptions of a variety of clinicians and academics who are interested in human nature and the human sciences.My aim in this commentary is to present an interpretation of a central finding of this questionnaire study: that psychologists were more likely than philosophers and scientists to embrace “psychophysical causation” as a part of their philosophical worldview. It is not, I think, too much of an oversimplification to define psychophysical causation as the presumption that mental events and physical events can cause other mental events and physical events. Psychophysical causation can be distinguished from other positions on the mind–body relation, some of which Fahrenberg and Cheetham mention in their article. Among these alternative positions is epiphenomenalism, the position that physical events cause other physical events and can cause mental events, but that mental events themselves have no causal power. Another philosophical position distinct from psychophysical causation is monism, which holds that all events in the world in general (and in human beings in particular) are physical and are best described in physical terms. Fahrenberg and Cheetham do not describe the multiple forms of monism in detail, but it is worth noting that reductive materialism (Wilson 1998) and eliminative materialism (Churchland 1981) are two highly developed forms of the kind of monism to which they allude. It is beyond the scope of this commentary to explore the differences between the two, but suffice it to say that both these forms of materialism reject the legitimacy of mental [End Page 205] events or the mental vocabulary that refers to such events.It is also beyond the scope of this commentary to advance arguments either for or against psychophysical causation in the philosophy of mind. The concept of psychophysical causation certainly has multiple proponents among philosophers, including those who are ontological monists. The compelling nonreductive materialism of a philosopher such as Terence Horgan, for example, suggests the viability of combining ontological monism with methodological pluralism. In a book chapter entitled “Nonreductive Materialism and the Explanatory Autonomy of Psychology,” Horgan (1993) developed the position that the world is entirely physical (i.e., there are no disembodied minds, persons, or other entities) but is so complex that it can only be understood and explained by employing many vocabularies and methodologies, spanning “the microphysical, neurobiological, macrobiological, and psychological.” This kind of methodological pluralism in the philosophy of mind can be disputed on conceptual grounds, but it seems to be at least as justifiable (and perhaps more justifiable) when compared with other well-defined positions on the mind–body relation.The appeal of nonreductive materialism and psychophysical causation comes into particularly clear focus in contexts where our philosophical concerns are pragmatic in nature. In other words, when we need to achieve practical goals in our everyday lives, we usually benefit from a philosophy that provides us with the flexibility to use a broad range of ideas, conceptual tools, and causal hypotheses that can be adapted to specific needs and challenges. Nonreductive materialism regards the world as a purely physical place, but ensures this kind of conceptual and methodological flexibility when it comes to explaining and predicting complex human experience and behavior. “Typically, certain context-relative features of discourse,” Horgan wrote (1993, 298), “will determine, in a given... (shrink)

Serious doubts about nonreductive materialism — the orthodoxy of the past two decades in philosophy of mind — have been long overdue. Jaegwon Kim has done perhaps the most to articulate the metaphysical problems that the new breed of materialists must confront in reconciling their physicalism with their commitment to the autonomy of the mental. Although the difficulties confronting supervenience, multiple-realizability, and mental causation have been recurring themes in his work, only mental causation — in particular, the (...) specter of epiphenomenalism — has really captured the interest of philosophers in general in recent years.This growing attention has spawned a large body of literature, which it is not our aim here to explore or assess. Rather, we want to call attention to what we believe is a new and quite different argumentative strategy against epiphenomenalism voiced in some recent articles by Tyler Burge and Stephen Yablo. Each has challenged two central assumptions of the existing mental causation debate. (shrink)

The analysis of dispositions is used to consider cases where the effect of one disposition operating is the existence of another disposition. This may arise from rearrangements within aggregated structures of dispositional parts, or, it is argued, also as stages of derivative dispositions within a set of multiple generative levels. Inspection of examples in both classical and quantum physics suggests a general principle of `Conditional Forward Causation': that dispositions act 'forwards' in a way conditional on certain circumstances or (...) occasions already existing at the `later' levels. (shrink)

Stephen Yablo has recently argued for a novel solution to the mental causation problem: the mental is related to the physical as determinables are related to determinates; determinables are not causal rivals with their determinates; so the mental and the physical are not causal rivals. Despite its attractions the suggestion seems hard to accept. In this paper I develop the idea that mental properties and physical properties are not causal rivals. Start with property dualism, supervenience, multiple realizability, and (...) the claim that no more than one supervenience base for a mental property can be had by a single instance of the mental property. Then a probabilistic account of causation will be unable to certify either mental properties or physical properties as causal factors for effect types. I suggest that this shows that we should not count mental properties as causal rivals with physical properties. (shrink)

The articles examines the teleofunctional solution to the problem of mental causation, presented by Dmitry Volkov in his recently published book Free Will. An Illusion or an Opportunity. D.B. Volkov proposes solutions to three big metaphysical problems – mental causation, personal identity, and free will. Solving the first problem, Volkov creatively combines the advantages of Dennett’s teleofunctional model and Vasilyev’s local interactionism. Volkov’s teleofunctional model of mental causation seeks to prove the causal relevance of mental properties as (...) non-local higher order properties. In my view, its substantiation is based on three points: (a) critics of the exclusion problem and Kim’s model of mental causation, (b) “Library of first editions” argument, (c) reduction of the causal trajectories argument (CTA 1) by Vasilyev to the counterpart argument (CTA 2) by Volkov. Each of these points faces objections. Kim’s criticism is based on an implicit confusion of two types of reduction – reduction from supervenience and from multiple realizability. The latter type does not threaten Kim’s ideas, but Volkov uses this very type in his criticism. The “Library of first editions” argument does not achieve its goal due to compositional features and because non-local relational properties are a type of external properties that cannot be causally relevant. The reduction of CTA 1 to CTA 2 is unsuccessful since, in the case of this reduction, important features of CTA 1 are lost – these are local mental properties, due to which the influence of non-local physical factors occurs. My main objection is that the concept of causally relevant non-local properties is incompatible with the very concept of cause. The set of causally relevant properties of cause can only be local. (shrink)

A theoretical framework is provided to explore teleonomy as a problem of self-causation, distinct from upward, downward and reticulate causation. Causality theories in biology are often formulated within hierarchy theories, where causation is conceptualized as running up or down the rungs of a ladder-like hierarchy or, more recently, as moving between multiple hierarchies. Research on the genealogy of cosmologies demonstrates that in addition to hierarchy theories, causality theories also depend upon ideas of time. This paper explores (...) the roots and impact of both time and hierarchy thinking on causal reasoning in the evolutionary sciences. Within evolutionary biology, the Neodarwinian synthesis adheres to a linear notion of time associated with linear hierarchies that portray upward causation. Eco-evo-devo schools recognize the importance of downward causation and consequently receive resistance from the standard view because downward causation is sometimes understood as backward causation, considered impossible by adherents of a linear time model. In contrast, downward causation works with a spatial or presential time notion. Hybridization, lateral gene transfer, infective heredity, symbiosis and symbiogenesis require recognition of reticulate causation occurring in both space and time, or spacetime, between distinct and interacting ontological hierarchies. Teleonomy is distinct from these types of causation because it invokes the problem of self-causation. By asking how the focal level in a hierarchy can persist through time, self-causation raises philosophical concerns on the nature of duration, identity and individuality. (shrink)

A statistical method for inference of formal causes was introduced. The procedure, referred to as the method of corresponding regressions, was explained and illustrated using a variety of simulated causal models. The method reflects IV/DV relations among variables traditionally limited to correlational or structural equation analysis. The method was applied to additive, subtractive, multiplicative, recursive and reflected models, as well as models of unrelated and correlated dependent variables. Initial applications to data from physical science, biology, economics, marketing and psychology were (...) developed, with generally supportive results. (shrink)

A close examination of Kim's argument in "Multiple Realization and the Metaphysics of Reduction" for the claim that if a kind is multiply realizable in a way that blocks identification with more fundamental properties it is also a kind unlikely to appear as an appropriate kind in a theory in the first place. Ultimately, I argue that there is one reasonably promising argument of this sort, but its success turns on explanatory questions the answers to which are far from (...) obvious. (shrink)

Many models of evolution are implicitly causal processes. Features such as causal feedback between evolutionary variables and evolutionary processes acting at multiple levels, though, mean that conventional causal models miss important phenomena. We develop here a general theoretical framework for analyzing evolutionary processes drawing on recent approaches to causal modeling developed in the machine-learning literature, which have extended Pearls do-calculus to incorporate cyclic causal interactions and multilevel causation. We also develop information-theoretic notions necessary to analyze causal information dynamics (...) in our framework, introducing a causal generalization of the Partial Information Decomposition framework. We show how our causal framework helps to clarify conceptual issues in the contexts of complex trait analysis and cancer genetics, including assigning variation in an observed trait to genetic, epigenetic and environmental sources in the presence of epigenetic and environmental feedback processes, and variation in fitness to mutation processes in cancer using a multilevel causal model respectively, as well as relating causally-induced to observed variation in these variables via information theoretic bounds. In the process, we introduce a general class of multilevel causal evolutionary processes which connect evolutionary processes at multiple levels via coarse-graining relationships. Further, we show how a range of fitness models can be formulated in our framework, as well as a causal analog of Prices equation, clarifying the relationships between realized/probabilistic fitness and direct/indirect selection. Finally, we consider the potential relevance of our framework to foundational issues in biology and evolution, including supervenience, multilevel selection and individuality. Particularly, we argue that our class of multilevel causal evolutionary processes, in conjunction with a minimum description length principle, provides a conceptual framework in which identification of multiple levels of selection may be reduced to a model selection problem. (shrink)

Top-down causation is often taken to be a metaphysically suspicious type of causation that is found in a few complex systems, such as in human mind-body relations. However, as Ellis and others have shown, top-down causation is ubiquitous in physics as well as in biology. Top-down causation occurs whenever specific dynamic behaviors are realized or selected among a broader set of possible lower-level states. Thus understood, the occurrence of dynamic and structural patterns in physical and biological (...) systems presents a problem for reductionist positions. We illustrate with examples of universality and functional equivalence classes how higher-level behaviors can be multiple realized by distinct lower-level systems or states. Multiple realizability in both contexts entails what Ellis calls “causal slack” between levels, or what others understand as relative explanatory autonomy. To clarify these notions further, we examine procedures for upscaling in multi-scale modeling. We argue that simple averaging strategies for upscaling only work for simplistic homogenous systems, because of the scale-dependency of characteristic behaviors in multi-scale systems. We suggest that this interpretation has implications for what Ellis calls mechanical top-down causation, as it presents a stronger challenge to reductionism than typically assumed. (shrink)

The causal closure of the physical poses a familiar causal exclusion problem for the special sciences that stems from the idea that if closure is true, then fundamental physical properties do all the causal work involved in bringing about physical effects. In this paper I aim to show that the strongest causal closure principle that is not ruled out by some simple physics in fact allows for a certain kind of downward causation, which in turn makes room for robust (...) special science autonomy. I focus on the case of vector composition, arguing that it involves irreducibly multilevel causation. To give a complete causal explanation of what goes on in cases that involve the effects of multiple forces, we need to appeal not only to the fundamental properties that generate those forces, but also to geometric properties that determine how multiple vectors compose. However, if all the causal work involved in such cases were due to fundamental physical properties, there would be a one-level explanation available. Hence, I argue, it cannot be the case that fundamental physical properties do all the causal work. The extra causal work, I suggest, comes from the fact that the causal powers bestowed by fundamental physical properties have irreducibly geometric manifestation conditions. I defend the resulting form of downward causation as a solution to the problem of special science autonomy, and discuss two potential accounts of its source, in terms of the debate between kinematic and dynamic theories of the origins of spacetime symmetries. (shrink)